Saturday, June 14, 2014

Rep. Rosa DeLauro (D-CT) Calls for Briefing on Beef Recalled for Mad Cow Potential Rep. Rosa DeLauro (D-CT)

UNITED STATES HOUSE OF REPRESENTATIVES ROSA L. DeLAURO COMMITTEE ON APPROPRIATIONS 3rd District, Connecticut

 

June 13,2014

 

The Honorable Mr. Brian Ronholm Acting Under Secretary for Food Safety U.S. Department of Agriculture 1400 Independence Ave.•SW Washington, DC 20250-3700

 

Dear Acting Under Secretary Ronholm,

 

I am deeply troubled by yesterday's news of the Fruitland American Meat establishment's recall of approximately 4,012 pounds of fresh beef products because of a potential risk for Bovine Spongiform Encephalopathy (BSE), also referred to as mad cow disease. This potentially tainted beef, "Rain Crow Ranch Ribeye," went to a Whole Foods distribution center in Connecticut that serves stores in New England and to two restaurants, one in New York City and one in Kansas City, Missouri.

 

My understanding is that the potentially tainted meat was not processed correctly for the age of these animals, resulting in parts of the nervous system that may not have been completely removed, which is not compliant with agency regulations that require their removal in cattle 30 months of age and older.

 

According to the FSIS news release the affected product was produced between September 5, 2013 through April 24, 2014. On the heels of all these other massive recalls, e.g. Wolverine, EI Rancho, etc., which also involved products produced over a long period of time, I question why it took ten months for this error to be discovered. Was this a matter of inadequate staffing levels, poor training, negligence, or some other reason? I want to learn more.

 

In view of this concern, I am requesting a briefing from you before June 27th, so that we can discuss these issues in depth. I look forward to meeting with you shortly.

 

Sincerely,

 

Rosa L. DeLauro Member of Congress

 


 

Greetings Honorable Rep. Rosa DeLauro Ma’am (D-CT) et al,

 

I wish to give you the information you requested above, but I will tell you now, nothing, NOTHING will happen. but thanks for trying anyway.

 

My mother and many more here in North America have been murdered by what I call corporate and political homicide, i.e. mad cow disease. she died from hvCJD i.e. the Heidenhain Variant of Creutzfeldt Jakob disease, and exceedingly rare strain of what officials call sporadic CJD. but what officials DON’t or WANT tell you, is that some cases of sporadic CJD have been linked to atypical BSE, and c-BSE i.e. typical UK type BSE, this will also propagate as sporadic CJD. this UKBSEnvCJD only theory is crap Ma’am, and proven to be so for trade purposes. I will not even get into mad deer disease aka CWD and risk factors there from here.

 

These SRM breaches, MBM mad cow feed breaches, BSE mad cow testing Breaches, Surveillance failures have happened time and time and time again over the past 16 years or so, and time and time again an official will raise awareness, and nothing happens. The GAO or the OIG issues warnings about these mad cow breaches, and NOTHING, nada. nothing will happen this time. For Gods sake, the USDA NSLP fed our children dead stock downer cows for 4 years, the most high risk for BSE TSE prion disease, before doing anything about it. I lost my mother to the hvCJD, confirmed, back in 1997, have watched and followed closely the USDA inc. mad cow follies over the past 16 years, and nothing has changed, except the administrations. we now have a human mad cow case now in Texas that the Government is covering up at all cost. the government refuses to issue any information on this case, except that the person traveled overseas, so it’s a foreign animal disease the man contracted...NOT!

 

fact is, BSE cases in Europe of the past years have dropped dramatically due to feed ban that was enforced, and extensive BSE testing, in large numbers. just the opposite has happened in the USA. it’s all been documented. there is ample evidence that there is as much of a chance (if not more), that this victim contracted human mad cow disease from sources right here in the USA. this PR push to alienate a USA source factor for human BSE in the USA is a PR stunt by the USDA inc., and not justified now, in my opinion. compare BSE testing figures in the EU compared to the USA, compare mad cow feed ban breaches, and you will see. hell, the 2004 enhanced BSE surveillance program was flawed so bad, the top Prion God at the NIH TSE prion expert Paul Brown, says he does not trust anything from the USDA since Texas covered up a mad cow for 7 months, on a 48 hour confirmation turn around. it’s all documented below in link. USDA inc shut down the mad cow testing after so many atypical BSE cases started showing up.

 

yes, another foreigner comes to the USA, or another USA citizens does some traveling, and all of a sudden, it’s a foreign disease. evidently, these folks never eat anything in the USA, and contracts nvcjd. right. just like the last one in 2005. really? here are the facts of the TEXAS MAD COW, MAD COW FEED in Texas, CJD CLUSTER in Texas, CJD CASE IN 38 YEAR OLD WOMAN THAT APPARENTLY WORKED ON THE SLAUGHTER LINE FOR TYSON in Texas, AND OTHER STRANGE TSE PRION DISEASE IN VERY YOUNG VICTIMS HERE IN TEXAS with long duration of illness from onset of clinical symptoms to death, CALLED SPORADIC FFI (except it is not linked to any genetic make up of that family), another nvcjd victim back in 2005 in Texas, apparently another UK victim that had moved to Texas, and never ate anything. these are the facts as I have come to know them (official documents), since hvcjd took my mom in December of 1997. just made a promise to mom, never forget, and never let them forget, the rest of the story, the truth you don’t hear about. ...our fine federal friends and the USDA inc, has lied to all of us...

 

 

HUMAN BSE NVCJD CASE IN TEXAS, COVER UP CONTINUES AS OF JUNE 14, 2014...TSS

 

 

Monday, June 02, 2014

 

*** Confirmed Human BSE aka mad cow Variant CJD vCJD or nvCJD Case in Texas

 


 

Monday, June 9, 2014

 

*** TEXAS MAD COW COVER UP (human BSE) AGAIN IN TEXAS, Mr. President Sir, we need your help please

 

Governor Rick Perry has done everything he can to cover up mad cow disease and human TSE prion disease there from in Texas over the last 15 years or so. We have another nvCJD case here in Texas i.e. human BSE, still no information there from, another lame excuse, typical though, and more junk science, we need help Mr. President...

 


 

Thursday, June 12, 2014

 

Missouri Firm Recalls Ribeye and Carcass Products That May Contain Specified Risk Materials 4,012 pounds of fresh beef products because the dorsal root ganglia may not have been completely removed

 


 

>>> FSIS and Fruitland American Meat have received no reports of adverse reactions due to consumption of these products.

 

>>> The USDA says there's no indication that the slaughtered cattle showed signs of BSE.

 

ha, ha, ha !

 

what a hoot.

 

our fine federal friends think we are all idiots. well, most of the lay public is oblivious to the science behind the TSE prion aka mad cow type disease.

 

SO, every time you here the FSIS, the USDA, the APHIS, or the FDA say that, ‘’NO REPORTED ILLNESS TO DATE’’, and or ‘’USDA says there's no indication that the slaughtered cattle showed signs of BSE’’, they are in fact calling every one of us idiots.

 

sadly, most of us go with the flow...and the industry keeps feeding us SRMs, and the government keeps telling us it’s all sporadic CJD.

 

and the world is happy $$$

 

the incubation period for the BSE CJD TSE prion is in YEARS TO DECADES. SUB-CLINICAL INFECTION IS A MAJOR RISK FACTOR !!!

 

Evidence of subclinical prion disease in aged mice following exposure to bovine spongiform encephalopathy

 

Karen L. Brown and Neil A. Mabbott

 

+ Author Affiliations The Roslin Institute and Royal (Dick) School of Veterinary Sciences, University of Edinburgh, Edinburgh, UK Correspondence Neil A. Mabbott neil.mabbott@roslin.ed.ac.uk Received 5 September 2013. Accepted 6 October 2013.

 

Abstract

 

The occurrence of variant Creutzfeldt–Jakob (vCJD) disease in humans was almost certainly the result of consumption of food contaminated with bovine spongiform encephalopathy (BSE) prions. Despite probable widespread exposure of the UK population to BSE-contaminated food in the 1980s, vCJD has been identified predominantly in young individuals, and there have been fewer cases of clinical disease than anticipated. The reasons for this are uncertain. Following peripheral exposure, many prions replicate within the lymphoid tissues before infecting the central nervous system. We have shown that the effects of host age on the microarchitecture of the spleen significantly impair susceptibility to mouse-adapted prions after peripheral exposure. The transmission of prions between different mammalian species is considered to be limited by the ‘species barrier’, which is dependent on several factors, including an intact immune system. Thus, cross-species prion transmission may be much less efficient in aged individuals. To test this hypothesis, we compared prion pathogenesis in groups of young (6–8 weeks old) and aged (600 days old) mice injected with primary BSE brain homogenate. We showed that prion pathogenesis was impaired dramatically in aged mice when compared with young animals. Whereas most young mice succumbed to clinical prion disease, all aged mice failed to develop clinical disease during their lifespans.

 

*** However, the demonstration that prion accumulation was detected in the lymphoid tissues of some aged mice after injection with primary BSE brain homogenate, in the absence of clinical signs of prion disease, has important implications for human health.

 


 

*** In some cases, the incubation period may be as long as 50 years

 


 

At a hearing in Parliament last Wednesday, the Science and Technology Committee was told that vCJD continued to pose a “significant” risk to UK public health and that more than one in every 2000 people could be silent carriers of the disease.

 

*** vCJD can have an incubation period of over 30 years.

 

Monday, February 03, 2014

 

CREUTZFELDT-JAKOB DISEASE T.S.E. PRION U.K. UPDATE As at 3rd February 2014

 


 

 

***In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures.

 


 

 

Hence, the data presented here are important for a risk- based SRM definition.

 

Competing interests

 

The authors declare that they have no competing interests.

 


 

see much more here ; Saturday, December 21, 2013

 

Complementary studies detecting classical bovine spongiform encephalopathy infectivity in jejunum, ileum and ileocaecal junction in incubating cattle

 


 

 

Friday, March 21, 2014

 

Rancho Dead Stock Cancer Downers Recall Explained FSIS March 20 2014 ?

 

“As of March 20, 2014, FSIS has completed all checks (effectiveness checks and disposition verification checks) for recalls 002-2014 and 013-2014 regarding Rancho Feeding Corporation. FSIS has determined that based on the number of successful checks (see Directive 8080.1, Attachment 1, Table 3) where businesses were notified of the recall and removed affected products from commerce that the recall activities were effective.”

 

hmmm ???

 


 

 

Thursday, November 28, 2013

 

Department of Justice Former Suppliers of Beef to National School Lunch Program Settle Allegations of Improper Practices and Mistreating Cows

 


 

*** seems USDA NSLP et al thought that it would be alright, to feed our children all across the USA, via the NSLP, DEAD STOCK DOWNER COWS, the most high risk cattle for mad cow type disease, and other dangerous pathogens, and they did this for 4 years, that was documented, then hid what they did by having a recall, one of the largest recalls ever, and they made this recall and masked the reason for the recall due to animal abuse (I do not condone animal abuse), not for the reason of the potential for these animals to have mad cow BSE type disease (or other dangerous and deadly pathogens). these TSE prion disease can lay dormant for 5, 10, 20 years, or longer, WHO WILL WATCH OUR CHILDREN FOR THE NEXT 5 DECADES FOR CJD ???

 

Saturday, September 21, 2013

 

Westland/Hallmark: 2008 Beef Recall A Case Study by The Food Industry Center January 2010 THE FLIM-FLAM REPORT

 


 

 

DID YOUR CHILD CONSUME SOME OF THESE DEAD STOCK DOWNER COWS, THE MOST HIGH RISK FOR MAD COW DISEASE ??? this recall was not for the welfare of the animals. ...tss you can check and see here ; (link now dead, does not work...tss)

 


 

 

try this link ;

 


 

 

 

FDA PART 589 -- SUBSTANCES PROHIBITED FROM USE IN ANIMAL FOOD OR FEED VIOLATIONS OFFICIAL ACTION INDICATED OAI UPDATE DECEMBER 2013 UPDATE

 

OAI 2012-2013

 

 OAI (Official Action Indicated) when inspectors find significant objectionable conditions or practices and believe that regulatory sanctions are warranted to address the establishment’s lack of compliance with the regulation. An example of an OAI classification would be findings of manufacturing procedures insufficient to ensure that ruminant feed is not contaminated with prohibited material. Inspectors will promptly re-inspect facilities classified OAI after regulatory sanctions have been applied to determine whether the corrective actions are adequate to address the objectionable conditions.

 

 ATL-DO 1035703 Newberry Feed & Farm Ctr, Inc. 2431 Vincent St. Newberry SC 29108-0714 OPR DR, FL, FR, TH HP 9/9/2013 OAI Y

 

 DET-DO 1824979 Hubbard Feeds, Inc. 135 Main, P.O. Box 156 Shipshewana IN 46565-0156 OPR DR, FL, OF DP 8/29/2013 OAI Y

 

 ATL-DO 3001460882 Talley Farms Feed Mill Inc 6309 Talley Rd Stanfield NC 28163-7617 OPR FL, TH NP 7/17/2013 OAI N

 

 NYK-DO 3010260624 Sherry Sammons 612 Stoner Trail Rd Fonda NY 12068-5007 OPR FR, OF NP 7/16/2013 OAI Y

 

 DEN-DO 3008575486 Rocky Ford Pet Foods 21693 Highway 50 East Rocky Ford CO 81067 OPR RE, TH HP 2/27/2013 OAI N

 

 CHI-DO 3007091297 Rancho Cantera 2866 N Sunnyside Rd Kent IL 61044-9605 OPR FR, OF HP 11/26/2012 OAI Y

 

 *** DEN-DO 1713202 Weld County Bi Products, Inc. 1138 N 11th Ave Greeley CO 80631-9501 OPR RE, TH HP 10/12/2012 OAI N

 

 Ruminant Feed Inspections Firms Inventory (excel format)

 


 

 PLEASE NOTE, the VAI violations were so numerous, and unorganized in dates posted, as in numerical order, you will have to sift through them for yourselves. ...tss

 

 Tuesday, June 11, 2013

 

 *** Weld County Bi-Products dba Fort Morgan Pet Foods 6/1/12 significant deviations from requirements in FDA regulations that are intended to reduce the risk of bovine spongiform encephalopathy (BSE) within the United States

 


 

 Thursday, June 6, 2013

 

 BSE TSE PRION USDA FDA MAD COW FEED COMPLIANCE REPORT and NAI, OAI, and VAI ratings as at June 5, 2013

 

 Greetings,

 

 since our fine federal friends have decided not to give out any more reports on the USA breaches of the feed ban and surveillance etc. for the BSE TSE prion mad cow type disease in the USDA livestock, I thought I might attempt it. I swear, I just don’t understand the logic of the SSS policy, and that includes all of it. I assure you, it would be much easier, and probably better for the FDA and the USDA INC., if they would simply put some kind of report out for Pete’s sake, instead of me doing it after I get mad, because I am going to put it all out there. the truth.

 

 PLEASE BE ADVISED, any breach of any of the above classifications OAI, VAI, RTS, CAN lead to breaches into the feed BSE TSE prion protocols, and CAN lead to the eventual suspect tainted feed reaching livestock. please, if any USDA official out there disputes this, please explain then how they could not. paperwork errors can eventually lead to breaches of the BSE TSE prion mad cow feed ban reaching livestock, or contamination and exposure there from, as well.

 

 I would sure like to see the full reports of just these ;

 

 4018 CHI-DO 3007091297 Rancho Cantera 2866 N Sunnyside Rd Kent IL 61044-9605 OPR FR, OF HP 11/26/2012 OAI Y

 

 9367 3008575486 Rocky Ford Pet Foods 21693 Highway 50 East Rocky Ford CO 81067 OPR RE, TH HP 2/27/2013 OAI N

 

 9446 DEN-DO 1713202 Weld County Bi Products, Inc. 1138 N 11th Ave Greeley CO 80631-9501 OPR RE, TH HP 10/12/2012 OAI N

 

 9447 DEN-DO 3002857110 Weld County Bi-Products dba Fort Morgan Pet Foods 13553 County Road 19 Fort Morgan CO 80701-7506 OPR RE HP 12/7/2011 OAI N

 

 see full list of the fda mad cow bse feed follies, toward the bottom, after a short brief update on the mad cow bse follies, and our good friend Lester Crawford that was at the FDA.

 

 ALSO, I would kindly like to comment on this FDA BSE/Ruminant Feed Inspections Firms Inventory (excel format)4 format, for reporting these breaches of BSE TSE prion protocols, from the extensive mad cow feed ban warning letters the fda use to put out for each violations. simply put, this excel format sucks, and the FDA et al intentionally made it this difficult to follow the usda fda mad cow follies. this is an intentional format to make it as difficult as possible to follow these breaches of the mad cow TSE prion safety feed protocols. to have absolutely no chronological or numerical order, and to format such violations in a way that they are almost impossible to find, says a lot about just how far the FDA and our fine federal friends will go through to hide these continued violations of the BSE TSE prion mad cow feed ban, and any breaches of protocols there from. once again, the wolf guarding the henhouse $$$

 

 NAI = NO ACTION INDICATED

 

 OAI = OFFICIAL ACTION INDICATED

 

 VAI = VOLUNTARY ACTION INDICATED

 

 RTS = REFERRED TO STATE

 

 Inspections conducted by State and FDA investigators are classified to reflect the compliance status at the time of the inspection, based upon whether objectionable conditions were documented. Based on the conditions found, inspection results are recorded in one of three classifications:

 

 OAI (Official Action Indicated) when inspectors find significant objectionable conditions or practices and believe that regulatory sanctions are warranted to address the establishment’s lack of compliance with the regulation. An example of an OAI classification would be findings of manufacturing procedures insufficient to ensure that ruminant feed is not contaminated with prohibited material. Inspectors will promptly re-inspect facilities classified OAI after regulatory sanctions have been applied to determine whether the corrective actions are adequate to address the objectionable conditions.

 

 VAI (Voluntary Action Indicated) when inspectors find objectionable conditions or practices that do not meet the threshold of regulatory significance, but warrant an advisory to inform the establishment that inspectors found conditions or practices that should be voluntarily corrected. VAI violations are typically technical violations of the 1997 BSE Feed Rule. These violations include minor recordkeeping lapses or conditions involving non-ruminant feeds.

 

 NAI (No Action Indicated) when inspectors find no objectionable conditions or practices or, if they find objectionable conditions, those conditions are of a minor nature and do not justify further actions.

 


 

 when sound science was bought off by junk science, in regards to the BSE TSE prion mad cow type disease, by the USDA, CFIA, WHO, OIE, et al. $$$

 

 when the infamous, and fraudulently USDA, FSIS, APHIS, FDA, gold card was taken away that infamous day in December of 2003, all cards were off the table, it was time to change the science, and change they did. ...tss

 

 snip. ...please see full text ;

 

 Thursday, June 6, 2013

 

 BSE TSE PRION USDA FDA MAD COW FEED COMPLIANCE REPORT and NAI, OAI, and VAI ratings as at June 5, 2013

 


 

 IN A NUT SHELL ;

 

 (Adopted by the International Committee of the OIE on 23 May 2006)

 

 11. Information published by the OIE is derived from appropriate declarations made by the official Veterinary Services of Member Countries. The OIE is not responsible for inaccurate publication of country disease status based on inaccurate information or changes in epidemiological status or other significant events that were not promptly reported to the Central Bureau,

 


 

 Sunday, December 15, 2013

 

 *** FDA PART 589 -- SUBSTANCES PROHIBITED FROM USE IN ANIMAL FOOD OR FEED VIOLATIONS OFFICIAL ACTION INDICATED OAI UPDATE DECEMBER 2013 UPDATE

 


 

 Saturday, November 10, 2012

 

 Wisconsin Firm Recalls Beef Tongues That May Contain Specified Risk Materials Nov 9, 2012 WI Firm Recalls Beef Tongues

 


 

 Saturday, July 23, 2011

 

 CATTLE HEADS WITH TONSILS, BEEF TONGUES, SPINAL CORD, SPECIFIED RISK MATERIALS (SRM's) AND PRIONS, AKA MAD COW DISEASE

 


 

 Sunday, October 18, 2009

 

 Wisconsin Firm Recalls Beef Tongues That Contain Prohibited Materials SRM WASHINGTON, October 17, 2009

 


 

 Thursday, October 15, 2009

 

 Nebraska Firm Recalls Beef Tongues That Contain Prohibited Materials SRM WASHINGTON, Oct 15, 2009

 


 

 Thursday, June 26, 2008

 

 Texas Firm Recalls Cattle Heads That Contain Prohibited Materials

 


 

 Tuesday, July 1, 2008

 

 Missouri Firm Recalls Cattle Heads That Contain Prohibited Materials SRMs

 


 

 Friday, August 8, 2008

 

 Texas Firm Recalls Cattle Heads That Contain Prohibited Materials SRMs 941,271 pounds with tonsils not completely removed

 


 

 Saturday, April 5, 2008

 

 SRM MAD COW RECALL 406 THOUSAND POUNDS CATTLE HEADS WITH TONSILS KANSAS

 


 

 Wednesday, April 30, 2008

 

 Consumption of beef tongue: Human BSE risk associated with exposure to lymphoid tissue in bovine tongue in consideration of new research findings

 


 

 Wednesday, April 30, 2008

 

 Consumption of beef tongue: Human BSE risk associated with exposure to lymphoid tissue in bovine tongue in consideration of new research findings

 


 

 Friday, October 15, 2010

 

 BSE infectivity in the absence of detectable PrPSc accumulation in the tongue and nasal mucosa of terminally diseased cattle

 


 

 SPECIFIED RISK MATERIALS SRMs

 


 

 2007

 

 10,000,000+ LBS. of PROHIBITED BANNED MAD COW FEED I.E. BLOOD LACED MBM IN COMMERCE USA 2007

 

 Date: March 21, 2007 at 2:27 pm PST

 

 RECALLS AND FIELD CORRECTIONS: VETERINARY MEDICINES -- CLASS II

 

 PRODUCT

 

 Bulk cattle feed made with recalled Darling's 85% Blood Meal, Flash Dried, Recall # V-024-2007

 

 CODE

 

 Cattle feed delivered between 01/12/2007 and 01/26/2007

 

 RECALLING FIRM/MANUFACTURER

 

 Pfeiffer, Arno, Inc, Greenbush, WI. by conversation on February 5, 2007.

 

 Firm initiated recall is ongoing.

 

 REASON

 

 Blood meal used to make cattle feed was recalled because it was cross- contaminated with prohibited bovine meat and bone meal that had been manufactured on common equipment and labeling did not bear cautionary BSE statement.

 

 VOLUME OF PRODUCT IN COMMERCE

 

 42,090 lbs.

 

 DISTRIBUTION

 

 WI

 

 ___________________________________

 

 PRODUCT

 

 Custom dairy premix products:

 

 MNM ALL PURPOSE Pellet, HILLSIDE/CDL Prot- Buffer Meal, LEE, M.-CLOSE UP PX Pellet, HIGH DESERT/ GHC LACT Meal, TATARKA, M CUST PROT Meal, SUNRIDGE/CDL PROTEIN Blend, LOURENZO, K PVM DAIRY Meal, DOUBLE B DAIRY/GHC LAC Mineral, WEST PIONT/GHC CLOSEUP Mineral, WEST POINT/GHC LACT Meal, JENKS, J/COMPASS PROTEIN Meal, COPPINI - 8# SPECIAL DAIRY Mix, GULICK, L-LACT Meal (Bulk), TRIPLE J - PROTEIN/LACTATION, ROCK CREEK/GHC MILK Mineral, BETTENCOURT/GHC S.SIDE MK-MN, BETTENCOURT #1/GHC MILK MINR, V&C DAIRY/GHC LACT Meal, VEENSTRA, F/GHC LACT Meal, SMUTNY, A- BYPASS ML W/SMARTA, Recall # V-025-2007

 

 CODE

 

 The firm does not utilize a code - only shipping documentation with commodity and weights identified.

 

 RECALLING FIRM/MANUFACTURER

 

 Rangen, Inc, Buhl, ID, by letters on February 13 and 14, 2007. Firm initiated recall is complete.

 

 REASON

 

 Products manufactured from bulk feed containing blood meal that was cross contaminated with prohibited meat and bone meal and the labeling did not bear cautionary BSE statement.

 

 VOLUME OF PRODUCT IN COMMERCE

 

 9,997,976 lbs.

 

 DISTRIBUTION

 

 ID and NV

 

 END OF ENFORCEMENT REPORT FOR MARCH 21, 2007

 


 

 Saturday, August 4, 2012

 

 Final Feed Investigation Summary - California BSE Case - July 2012

 


 

 Saturday, August 4, 2012

 

 Update from APHIS Regarding Release of the Final Report on the BSE Epidemiological Investigation

 


 

 Wednesday, September 25, 2013

 

 Inspections, Compliance, Enforcement, and Criminal Investigations BSE TSE PRION 2013

 


 

 Wednesday, October 30, 2013

 

 SPECIFIED RISK MATERIAL (SRM) CONTROL VERIFICATION TASK FSIS NOTICE 70-13 10/30/13

 


 

 Owens, Julie

 

 From: Terry S. Singeltary Sr. [flounder9@verizon.net]

 

 Sent: Monday, July 24, 2006 1:09 PM

 

 To: FSIS RegulationsComments

 

 Subject: [Docket No. FSIS-2006-0011] FSIS Harvard Risk Assessment of Bovine Spongiform Encephalopathy (BSE) Page 1 of 98

 


 

 FSIS, USDA, REPLY TO SINGELTARY

 


 

 2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006

 


 

remember, the sporadic cjd i.e. 85%+ of all human tse prion disease, has now been linked to ;

 

-------- Original Message --------

 

Subject: re-BSE prions propagate as either variant CJD-like or sporadic CJD

 

Date: Thu, 28 Nov 2002 10:23:43 -0000

 

From: "Asante, Emmanuel A" e.asante@ic.ac.uk

 

To: "'flounder@wt.net'" flounder@wt.net

 

Dear Terry,

 

I have been asked by Professor Collinge to respond to your request. I am a Senior Scientist in the MRC Prion Unit and the lead author on the paper. I have attached a pdf copy of the paper for your attention.

 

Thank you for your interest in the paper.

 

In respect of your first question, the simple answer is, ***yes. As you will find in the paper, we have managed to associate the alternate phenotype to type 2 PrPSc, the commonest sporadic CJD. It is too early to be able to claim any further sub-classification in respect of Heidenhain variant CJD or Vicky Rimmer's version. It will take further studies, which are on-going, to establish if there are sub-types to our initial finding which we are now reporting. The main point of the paper is that, as well as leading to the expected new variant CJD phenotype, BSE transmission to the 129-methionine genotype can lead to an alternate phenotype which is indistinguishable from type 2 PrPSc.

 

I hope reading the paper will enlighten you more on the subject. If I can be of any further assistance please to not hesitate to ask. Best wishes.

 

Emmanuel Asante

 

<>

 

____________________________________

 

Dr. Emmanuel A Asante MRC Prion Unit & Neurogenetics Dept. Imperial College School of Medicine (St. Mary's) Norfolk Place, LONDON W2 1PG Tel: +44 (0)20 7594 3794 Fax: +44 (0)20 7706 3272 email: e.asante@ic.ac.uk (until 9/12/02) New e-mail: e.asante@prion.ucl.ac.uk (active from now)

 

____________________________________

 

Thursday, August 12, 2010

 

Seven main threats for the future linked to prions

 

First threat

 

The TSE road map defining the evolution of European policy for protection against prion diseases is based on a certain numbers of hypotheses some of which may turn out to be erroneous. In particular, a form of BSE (called atypical Bovine Spongiform Encephalopathy), recently identified by systematic testing in aged cattle without clinical signs, may be the origin of classical BSE and thus potentially constitute a reservoir, which may be impossible to eradicate if a sporadic origin is confirmed.

 

***Also, a link is suspected between atypical BSE and some apparently sporadic cases of Creutzfeldt-Jakob disease in humans. These atypical BSE cases constitute an unforeseen first threat that could sharply modify the European approach to prion diseases.

 

Second threat

 

snip...

 


 

Monday, October 10, 2011

 

EFSA Journal 2011 The European Response to BSE: A Success Story

 

snip...

 

EFSA and the European Centre for Disease Prevention and Control (ECDC) recently delivered a scientific opinion on any possible epidemiological or molecular association between TSEs in animals and humans (EFSA Panel on Biological Hazards (BIOHAZ) and ECDC, 2011). This opinion confirmed Classical BSE prions as the only TSE agents demonstrated to be zoonotic so far

 

*** but the possibility that a small proportion of human cases so far classified as "sporadic" CJD are of zoonotic origin could not be excluded. Moreover, transmission experiments to non-human primates suggest that some TSE agents in addition to Classical BSE prions in cattle (namely L-type Atypical BSE, Classical BSE in sheep, transmissible mink encephalopathy (TME) and chronic wasting disease (CWD) agents) might have zoonotic potential.

 

snip...

 


 


 

To date the OIE/WAHO assumes that the human and animal health standards set out in the BSE chapter for classical BSE (C-Type) applies to all forms of BSE which include the H-type and L-type atypical forms. This assumption is scientifically not completely justified and accumulating evidence suggests that this may in fact not be the case. Molecular characterization and the spatial distribution pattern of histopathologic lesions and immunohistochemistry (IHC) signals are used to identify and characterize atypical BSE. Both the L-type and H-type atypical cases display significant differences in the conformation and spatial accumulation of the disease associated prion protein (PrPSc) in brains of afflicted cattle. Transmission studies in bovine transgenic and wild type mouse models support that the atypical BSE types might be unique strains because they have different incubation times and lesion profiles when compared to C-type BSE. When L-type BSE was inoculated into ovine transgenic mice and Syrian hamster the resulting molecular fingerprint had changed, either in the first or a subsequent passage, from L-type into C-type BSE.

 

***In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures.

 


 

Monday, May 19, 2014

 

*** Variant CJD: 18 years of research and surveillance

 


 

Discussion: The C, L and H type BSE cases in Canada exhibit molecular characteristics similar to those described for classical and atypical BSE cases from Europe and Japan. *** This supports the theory that the importation of BSE contaminated feedstuff is the source of C-type BSE in Canada. *** It also suggests a similar cause or source for atypical BSE in these countries. ***

 

see page 176 of 201 pages...tss

 


 

*** Singeltary reply ; Molecular, Biochemical and Genetic Characteristics of BSE in Canada Singeltary reply ;

 


 

Sunday, November 13, 2011

 

*** California BSE mad cow beef recall, QFC, CJD, and dead stock downer livestock

 


 

 

Mad cow disease: Could it be here?

 

Man's stubborn crusade attracts experts' notice By Carol Christian | August 5, 2001

 


 

yes, cjd is popping up in more and more places it seems. I think in 55 year and older, it's now 1 in 9,000.

 

see ;

 

lifetime risk of developing sporadic CJD is about 1 in 30,000, jumps to 1 in 9,000 in 50 years of age and above

 

IN REALITY, sporadic CJD is 1 in 9,000 in 50 years of age and above, and that's with a inadequate or what I call passive surveillance system. see below ;

 

Dr. William Shulaw, a veterinarian with The Ohio State University extension service, is involved in a nationwide program to eradicate scrapie, the form of BSE found in sheep.

 

Shulaw said the chances of a person getting sporadic Creutzfeldt- Jakob disease is about one in a million. But that's the total population, infants, children, adults and the elderly.

 

Chances increase to one in 9,000 when the group is restricted to those age 50 and older.

 


 


 


 

Tuesday, March 11, 2014

 

Science and Technology Committee Oral evidence: Blood, tissue and organ screening, HC 990 Wednesday 5 March 2014 SPORADIC CJD

 

Actually, it is nearer 2 per million per year of the population will develop sporadic CJD, but your lifetime risk of developing sporadic CJD is about 1 in 30,000. So that has not really changed. When people talk about 1 per million, often they interpret that as thinking it is incredibly rare. They think they have a 1-in-a-million chance of developing this disease. You haven’t. You’ve got about a 1-in-30,000 chance of developing it.

 


 

USA 2007-2008 sporadic CJD statistics revised to 1 in 9,000 in ages 55 and older !

 

The statistical incidence of CJD cases in the United States has been revised to reflect that there is one case per 9000 in adults age 55 and older. Eighty-five percent of the cases are sporadic, meaning there is no known cause at present.

 


 

but, the real count on sporadic CJD TSE human prion is not known...tss

 

Friday, April 25, 2014

 

Accuracy of administrative diagnostic data for pathologically confirmed cases of Creutzfeldt-Jakob disease

 


 

Creutzfeldt-Jakob Disease CJD cases rising North America updated report August 2013

 

*** Creutzfeldt-Jakob Disease CJD cases rising North America with Canada seeing an extreme increase of 48% between 2008 and 2010 ***

 


 

Sunday, October 13, 2013

 

*** CJD TSE Prion Disease Cases in Texas by Year, 2003-2012

 


 

Tuesday, April 01, 2014

 

Questions linger in U.S. CJD cases 2005, and still do in 2014

 


 

 

 

 

layperson

 

 

just made a promise to mom, never forget, never let them forget...dod 12/14/97 confirmed hvCJD TSE prion disease...

 

 

Terry S. Singeltary Sr.

P.O. Box 42

Bacliff, Texas USA 77518


 

1 comment:

Unknown said...

thank you for this valuable information. I lost my mother to this disease in Dec. 2013. The dr.'s acted like this was a brand new disease but i have since learned that just isn't talked about. More like covered up. I am angry that more hasn't been done about finding a cure. Reading this has brought some relief, at least I don't feel so alone in how I feel and there is someone out there just as baffled and angry as me. I just feel so helpless, I wish there was something more that can be done other than airing a grievence.