Unnecessary precautions BSE MAD COW DISEASE Dr. William James FSIS VS Dr.
Linda Detwiler
William James
Dr. William James capped a 28-year career at USDA’s Food Safety &
Inspection Service (FSIS) as the agency’s chief veterinarian. During his career
in FSIS he worked in the offices of Field Operations, Policy, Science, and
International Affairs. James supervised district offices, coordinated animal
welfare enforcement throughout the country, directed ante-mortem and post-mortem
inspection of livestock and poultry, implemented pathogen and residue sampling
and had executive oversight of import and export issues for FSIS.
Unnecessary precautions
(The views and opinions expressed in this blog are strictly those of the
author.)
Snuffy was the kids’ dog. Grew up with them. A beagle, so he was always
hungry.
Worst decision of my life was letting the kids make a house dog of him. No
food was safe around that ravenous animal. Good thing he was short. If you made
the mistake of setting your plate or bowl where he could reach it, you picked it
up empty.
The kids grew up and moved out. Eventually, Snuffy moved on, too. Sixteen
years is a long life for a dog. At least, it seemed long to us.
Snuffy has been gone for two years now and we have no other pets, but we
still can’t bring ourselves to set a plate or bowl on the floor by the couch or
leave any remnants of food where they might be reached by an ever-hungry beagle.
They’re unnecessary precautions, but we can’t seem to let go of them.
Changing gears a bit, many of you remember the terrible news when the first
case of Bovine Spongiform Encephalopathy (BSE) was diagnosed in the United
States. The news was released on December 23rd, 2003 and life for the beef
industry in this country changed for the worse.
In a matter of weeks, the Food Safety and Inspection Service (FSIS) issued
an Emergency Final Rule establishing various tissues in cattle of particular
ages as “specified risk materials,” or SRMs. SRMs are recognized as potential
sources of infectivity for BSE, and should be condemned in countries with BSE.
The rule was published in 2004. Ten years ago.
But the situation is different now. Due to the diligence of government and
industry both before and after the index case, BSE has never been able to gain a
foothold in this country. In May of last year, the World Organization for Animal
Health (OIE) declared the U.S. to be at “negligible” risk for BSE. “Negligible”
risk is the OIE’s best classification for the disease. It’s an important
upgrade.
Our national beef nightmare is over, right? Not entirely.
FSIS continues to require unnecessary precautions related to BSE. The
Agency announced in a Federal Register Notice on Jan. 13 its intention to renew
information collection about SRM management by federally inspected
establishments. As you already know, SRM removal is required by beef plants.
Companies must also have written plans and create records regarding removal and
disposal of SRMs. FSIS has no plan to rescind the SRM regulations.
Although FSIS is hanging on to the SRM regulations, the OIE does not
require these precautions to maintain the designation of “negligible” risk, or
to trade in beef from “negligible” risk countries. Also, the Animal and Plant
Health Inspection Service published a progressive final rule last November
bringing the United States in line with reasonable international trading norms
according to OIE risk guidelines.
It doesn’t help our export negotiations when we try to convince another
country to accept U.S. beef according to international standards, but we don’t
trust our own risk evaluation. Importantly, there has never been a recorded case
of the human form of BSE (variant Creutzfeldt-Jakob Disease) acquired in the
United State. Review of past cases of Alzheimer’s Disease by the Centers for
Disease Control and Prevention supports this assertion.
But FSIS can’t seem to let go of its precautions.
The rule is costly for both processors and taxpayers. It has no measurable
public health benefit. By definition, there is negligible risk. It’s time for
FSIS to stop acting like the James household and just move on.
I still miss that pesky dog.
2/12/2014
*********
Response from Linda Detwiler, Assistant Director of the Center for Public
and Corporate Veterinary Medicine (CPCVM) at the Virginia-Maryland Regional
College of Veterinary Medicine, University of Maryland:
I respectfully disagree with your assessment that the current BSE SRM
requirements are not necessary. Unlike your cute hound analogy where you have
definitive proof of zero risk, one cannot say the same about BSE. I could
provide > 5 pages of scientific justification for these requirements but I am
limited by the word count.
So in a nutshell:
• We do not truly know or understand the “real” risk to the public in
regards to vCJD as caused by classical BSE and any risk that may be caused by
atypical BSE.
• Per results of the UK’s appendix survey, it appears that humans may be
susceptible to infection with the BSE agent at doses below what was previously
considered. Currently the central prevalence estimate of vCJD in the UK is very
close to 1 in 2,000 in the age cohort tested.
• The UK has substantial evidence of “subclinical” vCJD and it is not known
whether these people will ever develop the disease and/or if they are a risk to
the blood or organ supply.
• We have less of an understanding of atypical BSE and its potential risk
to humans. All 3 native born cases in the US were atypical. The US surveillance
system is not intended to detect and eliminate such cases from the food supply,
hence after each case was reported, the government reassured the public that the
SRM bans were protective.
• In March 2013, APHIS will begin allowing the importation of certain live
bovines and bovine products from other countries. The risk to the US public is
not limited to the US system alone.
Comments on technical aspects of the risk assessment were then submitted to
FSIS. Comments were received from Food and Water Watch, Food Animal Concerns
Trust (FACT), Farm Sanctuary, R-CALF USA, Linda A Detwiler, and Terry S.
Singeltary. This document provides itemized replies to the public comments
received on the 2005 updated Harvard BSE risk assessment. Please bear the
following points in mind:
Owens, Julie From: Terry S. Singeltary Sr. [flounder9@verizon.net]
Sent: Monday, July 24, 2006 1:09 PM
To: FSIS RegulationsComments Subject: [Docket No. FSIS-2006-0011] FSIS
Harvard Risk Assessment of Bovine Spongiform Encephalopathy (BSE) Page 1 of 98
FSIS, USDA, REPLY TO SINGELTARY
U.S.A. 50 STATE BSE MAD COW CONFERENCE CALL Jan. 9, 2001
Tuesday, June 4, 2013
INTERPRETING RESULTS OF FSIS VERIFICATION SAMPLING OF DOMESTIC BEEF PRODUCT
DERIVED FROM ADVANCED MEAT RECOVERY SYSTEMS (AMR01/FAMR01) FSIS Notice 38-12
Thursday, May 30, 2013
World Organization for Animal Health (OIE) has upgraded the United States'
risk classification for mad cow disease to "negligible" from "controlled", and
risk further exposing the globe to the TSE prion mad cow type disease U.S. gets
top mad-cow rating from international group and risk further exposing the globe
to the TSE prion mad cow type disease
Tuesday, July 17, 2012
O.I.E. BSE, CWD, SCRAPIE, TSE PRION DISEASE Final Report of the 80th
General Session, 20 - 25 May 2012
IN SHORT, AND IN A NUT SHELL ;
(Adopted by the International Committee of the OIE on 23 May 2006) 11.
Information published by the OIE is derived from appropriate declarations made
by the official Veterinary Services of Member Countries. The OIE is not
responsible for inaccurate publication of country disease status based on
inaccurate information or changes in epidemiological status or other significant
events that were not promptly reported to the Central Bureau,
Friday, February 14, 2014
*** OFFAL from Class I Recall 002-2014 and 013-2014 Health Risk: High Jan
13, 2014 and Feb 8, 2014 shipped to Texas, Florida, and Illinois UPDATE FEBRUARY
14, 2014
Tuesday, February 11, 2014
*** California Firm Recalls Various Meat Products Produced Without the
Benefit of Full Inspection Class I Recall 002-2014 and 013-2014 Health Risk:
High Jan 13, 2014 and Feb 8, 2014
Wednesday, February 12, 2014
*** USDA/APHIS NOTICE: Final Rule Regarding Imports and BSE Effective March
4, 2014
Sunday, February 2, 2014
The Presence of Disease-Associated Prion Protein in Skeletal Muscle of
Cattle Infected with Classical Bovine Spongiform Encephalopathy
NOTE Pathology
*** October 2009 O.11.3
*** Infectivity in skeletal muscle of BASE-infected cattle
Silvia Suardi1, Chiara Vimercati1, Fabio Moda1, Ruggerone Margherita1,
Ilaria Campagnani1, Guerino Lombardi2, Daniela Gelmetti2, Martin H. Groschup3,
Anne Buschmann3, Cristina Casalone4, Maria Caramelli4, Salvatore Monaco5,
Gianluigi Zanusso5, Fabrizio Tagliavini1 1Carlo Besta" Neurological
Institute,Italy; 2IZS Brescia, Italy; 33FLI Insel Riems, D, Germany; 4CEA-IZS
Torino, Italy; 5University of Verona, Italy
Background: BASE is an atypical form of bovine spongiform encephalopathy
caused by a prion strain distinct from that of BSE. Upon experimental
transmission to cattle, BASE induces a previously unrecognized disease phenotype
marked by mental dullness and progressive atrophy of hind limb musculature.
Whether affected muscles contain infectivity is unknown. This is a critical
issue since the BASE strain is readily transmissible to a variety of hosts
including primates, suggesting that humans may be susceptible.
Objectives: To investigate the distribution of infectivity in peripheral
tissues of cattle experimentally infected with BASE. Methods: Groups of Tg mice
expressing bovine PrP (Tgbov XV, n= 7-15/group) were inoculated both i.c. and
i.p. with 10% homogenates of a variety of tissues including brain, spleen,
cervical lymph node, kidney and skeletal muscle (m. longissimus dorsi) from
cattle intracerebrally infected with BASE. No PrPres was detectable in the
peripheral tissues used for inoculation either by immunohistochemistry or
Western blot.
Results: Mice inoculated with BASE-brain homogenates showed clinical signs
of disease with incubation and survival times of 175±15 and 207±12 days. Five
out of seven mice challenged with skeletal muscle developed a similar
neurological disorder, with incubation and survival times of 380±11 and 410±12
days. At present (700 days after inoculation) mice challenged with the other
peripheral tissues are still healthy. The neuropathological phenotype and PrPres
type of the affected mice inoculated either with brain or muscle were
indistinguishable and matched those of Tgbov XV mice infected with natural BASE.
Discussion: Our data indicate that the skeletal muscle of cattle
experimentally infected with BASE contains significant amount of infectivity, at
variance with BSE-affected cattle, raising the issue of intraspecies
transmission and the potential risk for humans. Experiments are in progress to
assess the presence of infectivity in skeletal muscles of natural BASE.
1: J Infect Dis 1980 Aug;142(2):205-8
*** Oral transmission of kuru, Creutzfeldt-Jakob disease, and scrapie to
nonhuman primates.
Gibbs CJ Jr, Amyx HL, Bacote A, Masters CL, Gajdusek DC.
Kuru and Creutzfeldt-Jakob disease of humans and scrapie disease of sheep
and goats were transmitted to squirrel monkeys (Saimiri sciureus) that were
exposed to the infectious agents only by their nonforced consumption of known
infectious tissues. The asymptomatic incubation period in the one monkey exposed
to the virus of kuru was 36 months; that in the two monkeys exposed to the virus
of Creutzfeldt-Jakob disease was 23 and 27 months, respectively; and that in the
two monkeys exposed to the virus of scrapie was 25 and 32 months, respectively.
Careful physical examination of the buccal cavities of all of the monkeys failed
to reveal signs or oral lesions. One additional monkey similarly exposed to kuru
has remained asymptomatic during the 39 months that it has been under
observation.
snip...
The successful transmission of kuru, Creutzfeldt-Jakob disease, and scrapie
by natural feeding to squirrel monkeys that we have reported provides further
grounds for concern that scrapie-infected meat may occasionally give rise in
humans to Creutzfeldt-Jakob disease.
PMID: 6997404
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=6997404&dopt=Abstract
Sent: Sunday, December 15, 2013 8:15 PM
Subject: [BSE-L] FDA PART 589 -- SUBSTANCES PROHIBITED FROM USE IN ANIMAL
FOOD OR FEED VIOLATIONS OFFICIAL ACTION INDICATED OIA UPDATE DECEMBER 2013
UPDATE FDA PART 589 -- SUBSTANCES PROHIBITED FROM USE IN ANIMAL FOOD OR FEED
VIOLATIONS OFFICIAL ACTION INDICATED OIA UPDATE DECEMBER 2013
UPDATE OAI 2012-2013 OAI (Official Action Indicated) when inspectors find
significant objectionable conditions or practices and believe that regulatory
sanctions are warranted to address the establishment’s lack of compliance with
the regulation. An example of an OAI classification would be findings of
manufacturing procedures insufficient to ensure that ruminant feed is not
contaminated with prohibited material. Inspectors will promptly re-inspect
facilities classified OAI after regulatory sanctions have been applied to
determine whether the corrective actions are adequate to address the
objectionable conditions.
ATL-DO 1035703 Newberry Feed & Farm Ctr, Inc. 2431 Vincent St. Newberry
SC 29108-0714 OPR DR, FL, FR, TH HP 9/9/2013 OAI Y
DET-DO 1824979 Hubbard Feeds, Inc. 135 Main, P.O. Box 156 Shipshewana IN
46565-0156 OPR DR, FL, OF DP 8/29/2013 OAI Y
ATL-DO 3001460882 Talley Farms Feed Mill Inc 6309 Talley Rd Stanfield NC
28163-7617 OPR FL, TH NP 7/17/2013 OAI N
NYK-DO 3010260624 Sherry Sammons 612 Stoner Trail Rd Fonda NY 12068-5007
OPR FR, OF NP 7/16/2013 OAI Y
DEN-DO 3008575486 Rocky Ford Pet Foods 21693 Highway 50 East Rocky Ford CO
81067 OPR RE, TH HP 2/27/2013 OAI N
CHI-DO 3007091297 Rancho Cantera 2866 N Sunnyside Rd Kent IL 61044-9605 OPR
FR, OF HP 11/26/2012 OAI Y
*** DEN-DO 1713202 Weld County Bi Products, Inc. 1138 N 11th Ave Greeley CO
80631-9501 OPR RE, TH HP 10/12/2012 OAI N
Ruminant Feed Inspections Firms Inventory (excel format)
PLEASE NOTE, the VAI violations were so numerous, and unorganized in dates
posted, as in numerical order, you will have to sift through them for
yourselves. ...tss
SNIP...
SEE FULL TEXT ;
Sunday, December 15, 2013 FDA PART 589 -- SUBSTANCES PROHIBITED FROM USE IN
ANIMAL FOOD OR FEED VIOLATIONS OFFICIAL ACTION INDICATED OIA UPDATE DECEMBER
2013 UPDATE
I applaud the great efforts of the HSUS on their undercover work on this
and other cases, but in my opinion, USDA ET AL are wrong on the science of BSE
and risk factor there from from young animals, especially with veal (*** see BSE
Inquiry CJD report on veal and BSE), and the department of justice missed the
boat on the NSLP deadstock downer cow program, where for 4 years, the NSLP fed
deadstock downer cows to our children all across the Nation, the most high risk
cattle for mad cow disease and other deadly pathogens, then hid this fact under
a recall for animal abuse...these are the sad facts as I have come to know
them...who will watch our children for the next 50 years $$$
============================================================================================================
*** Individuals reported to eat veal on average at least once a year appear
to be at 13 TIMES THE RISK of individuals who have never eaten veal.
*** There is, however, a very wide confidence interval around this
estimate. There is no strong evidence that eating veal less than once per year
is associated with increased risk of CJD (p = 0.51).
*** The association between venison eating and risk of CJD shows similar
pattern, with regular venison eating associated with a 9 FOLD INCREASE IN RISK
OF CJD (p = 0.04).
*** There is some evidence that risk of CJD INCREASES WITH INCREASING
FREQUENCY OF LAMB EATING (p = 0.02). *** In conclusion, an analysis of dietary
histories revealed statistical associations between various meats/animal
products and INCREASED RISK OF CJD. When some account was taken of possible
confounding, the association between VEAL EATING AND RISK OF CJD EMERGED AS THE
STRONGEST OF THESE ASSOCIATIONS STATISTICALLY. ...
=============================================================================================================
CREUTZFELDT JAKOB DISEASE SURVEILLANCE IN THE UNITED KINGDOM THIRD ANNUAL
REPORT AUGUST 1994
Consumption of venison and veal was much less widespread among both cases
and controls. For both of these meats there was evidence of a trend with
increasing frequency of consumption being associated with increasing risk of
CJD. (not nvCJD, but sporadic CJD...tss) These associations were largely
unchanged when attention was restricted to pairs with data obtained from
relatives. ...
Table 9 presents the results of an analysis of these data.
*** There is STRONG evidence of an association between ‘’regular’’ veal
eating and risk of CJD (p = .0.01).
*** Individuals reported to eat veal on average at least once a year appear
to be at 13 TIMES THE RISK of individuals who have never eaten veal.
There is, however, a very wide confidence interval around this estimate.
There is no strong evidence that eating veal less than once per year is
associated with increased risk of CJD (p = 0.51).
The association between venison eating and risk of CJD shows similar
pattern, with regular venison eating associated with a 9 FOLD INCREASE IN RISK
OF CJD (p = 0.04).
There is some evidence that risk of CJD INCREASES WITH INCREASING FREQUENCY
OF LAMB EATING (p = 0.02).
The evidence for such an association between beef eating and CJD is weaker
(p = 0.14). When only controls for whom a relative was interviewed are included,
this evidence becomes a little STRONGER (p = 0.08).
snip...
It was found that when veal was included in the model with another
exposure, the association between veal and CJD remained statistically
significant (p = < 0.05 for all exposures), while the other exposures ceased
to be statistically significant (p = > 0.05).
snip...
In conclusion, an analysis of dietary histories revealed statistical
associations between various meats/animal products and INCREASED RISK OF CJD.
When some account was taken of possible confounding, the association between
VEAL EATING AND RISK OF CJD EMERGED AS THE STRONGEST OF THESE ASSOCIATIONS
STATISTICALLY. ...
snip...
In the study in the USA, a range of foodstuffs were associated with an
increased risk of CJD, including liver consumption which was associated with an
apparent SIX-FOLD INCREASE IN THE RISK OF CJD. By comparing the data from 3
studies in relation to this particular dietary factor, the risk of liver
consumption became non-significant with an odds ratio of 1.2 (PERSONAL
COMMUNICATION, PROFESSOR A. HOFMAN. ERASMUS UNIVERSITY, ROTTERDAM). (???...TSS)
snip...see full report ;
http://collections.europarchive.org/tna/20090505194948/http://bseinquiry.gov.uk/files/yb/1994/08/00004001.pdf
Thursday, October 10, 2013
*** CJD REPORT 1994 increased risk for consumption of veal and venison and
lamb
Thursday, November 28, 2013
Department of Justice Former Suppliers of Beef to National School Lunch
Program Settle Allegations of Improper Practices and Mistreating Cows
seems USDA NSLP et al thought that it would be alright, to feed our
children all across the USA, via the NSLP, DEAD STOCK DOWNER COWS, the most high
risk cattle for mad cow type disease, and other dangerous pathogens, and they
did this for 4 years, that was documented, then hid what they did by having a
recall, one of the largest recalls ever, and they made this recall and masked
the reason for the recall due to animal abuse (I do not condone animal abuse),
not for the reason of the potential for these animals to have mad cow BSE type
disease (or other dangerous and deadly pathogens). these TSE prion disease can
lay dormant for 5, 10, 20 years, or longer, WHO WILL WATCH OUR CHILDREN FOR THE
NEXT 5 DECADES FOR CJD ???
Saturday, September 21, 2013
Westland/Hallmark: 2008 Beef Recall A Case Study by The Food Industry
Center January 2010 THE FLIM-FLAM REPORT
DID YOUR CHILD CONSUME SOME OF THESE DEAD STOCK DOWNER COWS, THE MOST HIGH
RISK FOR MAD COW DISEASE ??? this recall was not for the welfare of the animals.
...tss you can check and see here ; (link now dead, does not work...tss)
try this link ;
Sunday, November 13, 2011
California BSE mad cow beef recall, QFC, CJD, and dead stock downer
livestock
Thursday, February 13, 2014
HSUS VS USDA ET AL BAN DOWNER CALVES FOR HUMAN CONSUMPTION (*veal) and
potential BSE risk factor there from
*** see today ;
> > > The CWD infection rate was nearly 80%, the highest ever in a
North American captive herd. <<<
From: Terry S. Singeltary Sr.
Sent: Tuesday, December 20, 2011 10:18 PM
Subject: [BSE-L] CHRONIC WASTING DISEASE CWD WISCONSIN Almond Deer
(Buckhorn Flats) Farm Update DECEMBER 2011
CHRONIC WASTING DISEASE CWD WISCONSIN Almond Deer (Buckhorn Flats) Farm
Update DECEMBER 2011
SNIP...SEE FULL TEXT ;
Wednesday, September 04, 2013
***cwd - cervid captive livestock escapes, loose and on the run in the
wild...
2014 CWD UPDATE
*** PRICE OF CWD TSE PRION POKER GOES UP 2014 ***
Transmissible Spongiform Encephalopathy TSE PRION update January 2, 2014
*** chronic wasting disease, there was no absolute barrier to conversion of
the human prion protein.
*** Furthermore, the form of human PrPres produced in this in vitro assay
when seeded with CWD, resembles that found in the most common human prion
disease, namely sCJD of the MM1 subtype.
Wednesday, January 01, 2014
Molecular Barriers to Zoonotic Transmission of Prions
*** chronic wasting disease, there was no absolute barrier to conversion of
the human prion protein.
*** Furthermore, the form of human PrPres produced in this in vitro assay
when seeded with CWD, resembles that found in the most common human prion
disease, namely sCJD of the MM1 subtype.
*** The potential impact of prion diseases on human health was greatly
magnified by the recognition that interspecies transfer of BSE to humans by beef
ingestion resulted in vCJD. While changes in animal feed constituents and
slaughter practices appear to have curtailed vCJD, there is concern that CWD of
free-ranging deer and elk in the U.S. might also cross the species barrier.
Thus, consuming venison could be a source of human prion disease. Whether BSE
and CWD represent interspecies scrapie transfer or are newly arisen prion
diseases is unknown. Therefore, the possibility of transmission of prion disease
through other food animals cannot be ruled out. There is evidence that vCJD can
be transmitted through blood transfusion. There is likely a pool of unknown size
of asymptomatic individuals infected with vCJD, and there may be asymptomatic
individuals infected with the CWD equivalent. These circumstances represent a
potential threat to blood, blood products, and plasma supplies.
Thursday, January 2, 2014
*** CWD TSE Prion in cervids to hTGmice, Heidenhain Variant
Creutzfeldt-Jacob Disease MM1 genotype, and iatrogenic CJD ??? ***
SNIP...
Subtype 1: (sCJDMM1 and sCJDMV1)
This subtype is observed in patients who are MM homozygous or MV
heterozygous at codon 129 of the PrP gene (PRNP) and carry PrPSc Type 1.
Clinical duration is short, 3‑4 months.32 The most common presentation in
sCJDMM1 patients is cognitive impairment leading to frank dementia, gait or limb
ataxia, myoclonic jerks and visual signs leading to cortical blindness
(Heidenhain’s syndrome)...
Animals injected with iatrogenic Creutzfeldt–Jakob disease MM1 and genetic
Creutzfeldt–Jakob disease MM1 linked to the E200K mutation showed the same
phenotypic features as those infected with sporadic Creutzfeldt–Jakob disease
MM1 prions...
*** our results raise the possibility that CJD cases classified as VV1 may
include cases caused by iatrogenic transmission of sCJD-MM1 prions or food-borne
infection by type 1 prions from animals, e.g., chronic wasting disease prions in
cervid. In fact, two CJD-VV1 patients who hunted deer or consumed venison have
been reported (40, 41). The results of the present study emphasize the need for
traceback studies and careful re-examination of the biochemical properties of
sCJD-VV1 prions. ***
SNIP...SEE FULL TEXT ;
Thursday, January 2, 2014
*** CWD TSE Prion in cervids to hTGmice, Heidenhain Variant
Creutzfeldt-Jacob Disease MM1 genotype, and iatrogenic CJD ??? ***
Wednesday, January 01, 2014
APHIS-2006-0118-0100 Chronic Wasting Disease Herd Certification Program and
Interstate Movement of Farmed or Captive Deer, Elk, and Moose
Friday, November 22, 2013
Wasting disease is threat to the entire UK deer population CWD TSE Prion
disease Singeltary submission to Scottish Parliament
Monday, February 3, 2014
*** Evaluation of the zoonotic potential of transmissible mink
encephalopathy TSE Prion disease
Research Project: TRANSMISSION, DIFFERENTIATION, AND PATHOBIOLOGY OF
TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHIES
Sunday, February 16, 2014
Recycling collection to benefit young mother with rare, terminal
disease
Buy This Photo Photo 1 of 1 | Zoom Photo + Top Photo Sandi Kennedy, shown
here with her oldest son, is now home with family and the community is rallying
around her. Courtesy photo By Staff reports February 14, 2014 3:21 PM KENNEBUNK
- The CAN DO program at the Kennebunk Transfer Station will spend the next two
months collecting cans and bottles to help the family of Sandi Kennedy, a young
Kennebunk mother struck by a rare, fatal disease.
"Drop off bottles and cans with a redemption value (Maine State deposit
only) at the Transfer Station on Sea Road," said CAN DO organizer Tom Couming.
"This will continue for at least two months."
Doctors have diagnosed Kennedy, a 38-year-old wife and mom of four young
children ages 2 to 9, with Creutzfeldt-Jakob Disease, an incurable neurological
disorder, part of a family known as prion diseases.
Kennedy is now at her home in Kennebunk, surrounded by family, and will be
on hospice care as doctors have given her a short time to live.
Friends and family members have created a page on YouCaring.com to support
the Kennedy family. Visit youcaring.com and search for "Hope for Sandi." One
hundred percent of any money raised through the page will benefit the
Kennedys.
Sunday, August 09, 2009
CJD...Straight talk with...James Ironside...and...Terry Singeltary... 2009
Friday, January 10, 2014
vpspr, sgss, sffi, TSE, an iatrogenic by-product of gss, ffi, familial type
prion disease, what it ???
Monday, February 03, 2014
CREUTZFELDT-JAKOB DISEASE T.S.E. PRION U.K. UPDATE As at 3rd February 2014
WHAT about the sporadic CJD TSE proteins ?
WE now know that some cases of sporadic CJD are linked to atypical BSE and
atypical Scrapie, so why are not MORE concerned about the sporadic CJD, and all
it’s sub-types $$$
Creutzfeldt-Jakob Disease CJD cases rising North America updated report
August 2013
*** Creutzfeldt-Jakob Disease CJD cases rising North America with Canada
seeing an extreme increase of 48% between 2008 and 2010 ***
Sunday, October 13, 2013
*** CJD TSE Prion Disease Cases in Texas by Year, 2003-2012
Sunday, January 19, 2014
National Prion Disease Pathology Surveillance Center Cases Examined1 as of
January 8, 2014
Wednesday, December 11, 2013
*** Detection of Infectivity in Blood of Persons with Variant and Sporadic
Creutzfeldt-Jakob Disease ***
Friday, February 14, 2014
Creutzfeldt-Jakob disease (CJD) biannual update (February 2014), with
briefing on novel human prion disease National CJD Research and Surveillance
Unit NCJDRSU
TSS
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