Rancho Co-Owner Will Go To Trial Alone, Three Others Make Plea Deals
By Dan Flynn | December 19, 2014
Jury selection will begin July 16, 2015, in the federal criminal
conspiracy case involving former Rancho Feeding Corp. co-owner Jesse J. Amaral
Jr. The 76-year-old cattle company executive will be tried alone as three
others, implicated in the alleged conspiracy to sell for human consumption
cattle known to have cancerous eyeballs, have all made deals with the
prosecution.
Felix Sandoval Cabrera, 55, the foreman of Rancho’s slaughterhouse at
Petaluma, CA, is the latest to reach a plea agreement with the government,
entering a single guilty plea to count 7 of the original indictment last Aug. 14
charging him with distribution of adulterated, misbranded and uninspected meat.
Earlier, Eugene D. Corda, the 65-year-old Rancho yardman, and 77-year-old
Robert Singleton, Rancho’s other co-owner, also entered guilty pleas to the same
sole count.
That leaves only Amaral going to trial next July. The court-approved plea
agreements the other three defendants have with the government are sealed, but
it’s likely that all three have agreed to appear at the trial as government
witnesses against him.
The prosecution has charged the former Rancho co-owner with 11 federal
felony counts. The government will attempt to prove to a jury that the Petaluma
resident is guilty of one count of conspiracy to distribute adulterated,
misbranded and uninspected meat, two counts of conspiracy to commit mail fraud,
six counts of distribution of adulterated and misbranded meat, one count of
conspiracy to commit mail fraud, and two counts of mail fraud.
If convicted on all counts, Amaral could be sentenced to more than 100
years in jail and fined more than $1.3 million.
Proceedings for all the defendants are being held before U.S. District
Judge Charles R. Breyer in San Francisco. He has scheduled a status conference
for Singleton on Feb. 18, 2015, and another for Cabrera and Corda on Aug. 12,
2015, which will likely be after the Amaral trial has concluded.
Amaral, Rancho’s president and general manager, was in control of the
day-to-day operations at the Petaluma slaughterhouse located 60 miles north of
San Francisco. Singleton’s role was to buy cattle and supervise processed beef
for distribution.
Cabrera, Rancho’s foreman, was responsible for the staff and the “kill
floor,” including being responsible for “knocking cattle,” or stunning them
immediately prior to slaughter.
As yardman, Corda was responsible for receiving cattle and moving them to
the proper areas for inspection and slaughter.
Singleton bought cattle from both auction houses and individual farmers
and ranchers in Northern California and Nevada.
“Some of the purchased cattle exhibited signs of epithelioma, that is
lumps or other abnormalities around the eye, and were less expensive than cattle
that appeared completely healthy,” according to the indictment.
When cattle with these eye conditions arrived at the Petaluma
slaughterhouse, Corda or another Rancho employee would allegedly place them in
pen 9A, court documents state. The owners were in charge of determining the
order in which cattle were processed for inspection and slaughter.
When instructed, Corda moved cattle into the pen designated for
ante-mortem inspection by the USDA veterinarian or Food Safety and Inspection
Service (FSIS) personnel. Cattle passing ante-mortem inspection generally went
immediately into the kill chute, where they were knocked, slaughtered and
inspected again post mortem. After passing the post-mortem inspection, the
carcass was tagged and could be sold.
In mid-2012, Amaral is accused of ordering Rancho employees to process
cattle that were condemned by the USDA veterinarian. At his instruction, Cabrera
allegedly had workers cut the “USDA Condemned” stamps out of the cattle
carcasses so they could be processed for sale and distribution.
At about the same time, prosecutors say Amaral gave the foreman, Cabrera,
and the yardman, Corda, directions on how to circumvent inspection procedures
for cows with cancerous eyes. Both Amaral and Singleton allegedly told their
employees to swap out uninspected cows with cancerous eyes with cattle that had
already passed ante-mortem inspection.
“Cabrera knocked the cancer eye cows, and he or another kill floor
employee at his instruction slaughtered them and deposited their heads in the
gut bin,” the indictment states. “Cabrera, or another kill floor employee at his
instruction, placed heads from apparently healthy cows, which had been
previously reserved, next to the cancer eye cow carcasses.
“The switch and slaughter of un-inspected cancer eye cows occurred during
the inspectors’ lunch breaks, at a time during which plant operations were
supposed to cease,” the charging documents continue. “When the inspectors
returned from lunch for post mortem inspections, they were unaware that the
carcasses they were inspecting belonged to cancer eye cows that had escaped ante
mortem inspection.”
Based on Rancho’s records, the government found that, from January 2013 to
January 2014, beef from 101 head of condemned cattle and 79 cows with eye cancer
was processed for human consumption. For every diseased animal that he got past
USDA inspectors, Cabrera allegedly got a $50 bonus.
In early 2014, with the federal investigation underway, Rancho recalled
all of its beef production going back the previous year, or about 8.7 million
pounds. It also was forced to close down and ultimately sell the Petaluma
slaughterhouse. The facility has since resumed operations under new ownership.
© Food Safety News
More Headlines from Food Policy & Law »
Tags: Eugene D. Corda, Felix Sandoval Cabrera, Jesse J. Amaral Jr.,
Petaluma, Rancho, Rancho Feeding Corporation, Robert Singleton
INDICTMENT
see more on Rancho history here;
Friday, March 21, 2014
Rancho Dead Stock Cancer Downers Recall Explained FSIS March 20 2014
?
“As of March 20, 2014, FSIS has completed all checks (effectiveness checks
and disposition verification checks) for recalls 002-2014 and 013-2014 regarding
Rancho Feeding Corporation. FSIS has determined that based on the number of
successful checks (see Directive 8080.1, Attachment 1, Table 3) where businesses
were notified of the recall and removed affected products from commerce that the
recall activities were effective.”
Thursday, March 20, 2014
JACK IN THE BOX NOW CAUGHT UP IN MASSIVE RANCHO DEAD STOCK DOWNER CANCER
COW RECALL
Thursday, March 6, 2014
TEXAS RECALL LIST MASSIVE FROM DEAD STOCK DOWNER CANCER COWS OFFAL from
Class I Recall 002-2014 and 013-2014 Health Risk: High Jan 13, 2014 and Feb 8,
2014 shipped to Texas, Florida, and Illinois UPDATE FEBRUARY 14, 2014
Thursday, February 27, 2014
BEEF, CANCER, PRIONS, AND OTHER DANGEROUS AND DEADLY PATHOGENS, APPARENTLY,
IT'S WHAT'S FOR DINNER
>>>“Cabrera knocked the cancer eye cows, and he or another kill
floor employee at his instruction slaughtered them and deposited their heads in
the gut bin,” the indictment states. “Cabrera, or another kill floor employee at
his instruction, placed heads from apparently healthy cows, which had been
previously reserved, next to the cancer eye cow carcasses. <<<
where have I heard something similar before ?
Galen Niehues, an inspector for the Nebraska Department of
Agriculture, (NDA), was convicted of mail fraud for submitting falsified reports
to his employer concerning inspections he was supposed to perform at Nebraska
cattle operations. Niehues was tasked with performing inspections of Nebraska
ranches, cattle and feed for the presence of neurological diseases in cattle
including Bovine Spongiform Encephalopathy (BSE), also known as “Mad Cow
Disease”. Niehues was to identify cattle producers, perform on-site inspections
of the farm sites and cattle operations, ask producers specific questions about
feed, and take samples of the feed. Niehues was to then submit feed samples for
laboratory analysis, and complete reports of his inspections and submit them to
the NDA and to the Federal Food and Drug Administration (FDA). An investigation
by the FDA and NDA revealed Niehues had fabricated approximately 100 BSE
inspections and inspection reports. When confronted, Niehues admitted his
reports were fraudulent, and that had fabricated the reports and feed samples he
submitted to the NDA. Niehues received a sentence of 5 years probation, a 3-year
term of supervised release, and was required to pay $42,812.10 in
restitution.
Thursday, November 18, 2010
UNITED STATES OF AMERICA VS GALEN J. NIEHUES FAKED MAD COW FEED TEST ON 92
BSE INSPECTION REPORTS FOR APPROXIMATELY 100 CATTLE OPERATIONS
Date: June 21, 2007 at 2:49 pm PST
Owner and Corporation Plead Guilty to Defrauding Bovine Spongiform
Encephalopathy (BSE) Surveillance Program
An Arizona meat processing company and its owner pled guilty in February
2007 to charges of theft of Government funds, mail fraud, and wire fraud. The
owner and his company defrauded the BSE Surveillance Program when they falsified
BSE Surveillance Data Collection Forms and then submitted payment requests to
USDA for the services. In addition to the targeted sample population (those
cattle that were more than 30 months old or had other risk factors for BSE), the
owner submitted to USDA, or caused to be submitted, BSE obex (brain stem)
samples from healthy USDA-inspected cattle. As a result, the owner fraudulently
received approximately $390,000. Sentencing is scheduled for May 2007.
snip...
Topics that will be covered in ongoing or planned reviews under Goal 1
include:
soundness of BSE maintenance sampling (APHIS),
implementation of Performance-Based Inspection System enhancements for
specified risk material (SRM) violations and improved inspection controls over
SRMs (FSIS and APHIS),
snip...
The findings and recommendations from these efforts will be covered in
future semiannual reports as the relevant audits and investigations are
completed.
4 USDA OIG SEMIANNUAL REPORT TO CONGRESS FY 2007 1st Half
-MORE Office of the United States Attorney District of Arizona
FOR IMMEDIATE RELEASE For Information Contact Public Affairs
February 16, 2007 WYN HORNBUCKLE Telephone: (602) 514-7625 Cell: (602)
525-2681
CORPORATION AND ITS PRESIDENT PLEAD GUILTY TO DEFRAUDING GOVERNMENT’S MAD
COW DISEASE SURVEILLANCE PROGRAM
PHOENIX -- Farm Fresh Meats, Inc. and Roland Emerson Farabee, 55, of
Maricopa, Arizona, pleaded guilty to stealing $390,000 in government funds, mail
fraud and wire fraud, in federal district court in Phoenix. U.S. Attorney Daniel
Knauss stated, “The integrity of the system that tests for mad cow disease
relies upon the honest cooperation of enterprises like Farm Fresh Meats. Without
that honest cooperation, consumers both in the U.S. and internationally are at
risk. We want to thank the USDA’s Office of Inspector General for their
continuing efforts to safeguard the public health and enforce the law.” Farm
Fresh Meats and Farabee were charged by Information with theft of government
funds, mail fraud and wire fraud. According to the Information, on June 7, 2004,
Farabee, on behalf of Farm Fresh Meats, signed a contract with the U.S.
Department of Agriculture (the “USDA Agreement”) to collect obex samples from
cattle at high risk of mad cow disease (the “Targeted Cattle Population”). The
Targeted Cattle Population consisted of the following cattle: cattle over thirty
months of age; nonambulatory cattle; cattle exhibiting signs of central nervous
system disorders; cattle exhibiting signs of mad cow disease; and dead cattle.
Pursuant to the USDA Agreement, the USDA agreed to pay Farm Fresh Meats $150 per
obex sample for collecting obex samples from cattle within the Targeted Cattle
Population, and submitting the obex samples to a USDA laboratory for mad cow
disease testing. Farm Fresh Meats further agreed to maintain in cold storage the
sampled cattle carcasses and heads until the test results were received by Farm
Fresh Meats.
Evidence uncovered during the government’s investigation established that
Farm Fresh Meats and Farabee submitted samples from cattle outside the Targeted
Cattle Population. Specifically, Farm Fresh Meats and Farabee submitted, or
caused to be submitted, obex samples from healthy, USDA inspected cattle, in
order to steal government moneys.
Evidence collected also demonstrated that Farm Fresh Meats and Farabee
failed to maintain cattle carcasses and heads pending test results and falsified
corporate books and records to conceal their malfeasance. Such actions, to the
extent an obex sample tested positive (fortunately, none did), could have
jeopardized the USDA’s ability to identify the diseased animal and pinpoint its
place of origin. On Wednesday, February 14, 2007, Farm Fresh Meats and Farabee
pleaded guilty to stealing government funds and using the mails and wires to
effect the scheme. According to their guilty pleas:
(a) Farm Fresh Meats collected, and Farabee directed others to collect,
obex samples from cattle outside the Targeted Cattle Population, which were not
subject to payment by the USDA;
(b) Farm Fresh Meats 2 and Farabee caused to be submitted payment requests
to the USDA knowing that the requests were based on obex samples that were not
subject to payment under the USDA Agreement;
(c) Farm Fresh Meats completed and submitted, and Farabee directed others
to complete and submit, BSE Surveillance Data Collection Forms to the USDA’s
testing laboratory that were false and misleading;
(d) Farm Fresh Meats completed and submitted, and Farabee directed others
to complete and submit, BSE Surveillance Submission Forms filed with the USDA
that were false and misleading;
(e) Farm Fresh Meats falsified, and Farabee directed others to falsify,
internal Farm Fresh Meats documents to conceal the fact that Farm Fresh Meats
was seeking and obtaining payment from the USDA for obex samples obtained from
cattle outside the Targeted Cattle Population; and
(f) Farm Fresh Meats failed to comply with, and Farabee directed others to
fail to comply with, the USDA Agreement by discarding cattle carcasses and heads
prior to receiving BSE test results. A conviction for theft of government funds
carries a maximum penalty of 10 years imprisonment. Mail fraud and wire fraud
convictions carry a maximum penalty of 20 years imprisonment. Convictions for
the above referenced violations also carry a maximum fine of $250,000 for
individuals and $500,000 for organizations. In determining an actual sentence,
Judge Earl H. Carroll will consult the U.S. Sentencing Guidelines, which provide
appropriate sentencing ranges. The judge, however, is not bound by those
guidelines in determining a sentence.
Sentencing is set before Judge Earl H. Carroll on May 14, 2007. The
investigation in this case was conducted by Assistant Special Agent in Charge
Alejandro Quintero, United States Department of Agriculture, Office of Inspector
General. The prosecution is being handled by Robert Long, Assistant U.S.
Attorney, District of Arizona, Phoenix. CASE NUMBER: CR-07-00160-PHX-EHC RELEASE
NUMBER: 2007-051(Farabee) # # #
WE can only hope that this is a single incident. BUT i have my doubts. I
remember when the infamous TOKEN Purina Feed Mill in Texas was feeding up to 5.5
grams of potentially and probably tainted BANNED RUMINANT feed to cattle, and
the FDA was bragging at the time that the amount of potentially BANNED product
was so little and the cattle were so big ;
"It is important to note that the prohibited material was domestic in
origin (therefore not likely to contain infected material because there is no
evidence of BSE in U.S. cattle), fed at a very low level, and fed only once. The
potential risk of BSE to such cattle is therefore exceedingly low, even if the
feed were contaminated."
On Friday, April 30 th , the Food and Drug Administration learned that a
cow with central nervous system symptoms had been killed and shipped to a
processor for rendering into animal protein for use in animal feed. ... FDA's
investigation showed that the animal in question had already been rendered into
"meat and bone meal" (a type of protein animal feed). Over the weekend FDA was
able to track down all the implicated material. That material is being held by
the firm, which is cooperating fully with FDA.
WE now know all that was a lie. WE know that literally Thousands of TONS of
BANNED and most likely tainted product is still going out to commerce. WE know
now and we knew then that .005 to a gram was lethal. WE know that CWD infected
deer and elk, scrapie infected sheep, BSE and BASE infected cattle have all been
rendered and fed back to livestock (including cattle) for human and animal
consumption.
Paul Brown, known and respected TSE scientist, former TSE expert for the
CDC said he had ''absolutely no confidence in USDA tests before one year ago'',
and this was on March 15, 2006 ;
"The fact the Texas cow showed up fairly clearly implied the existence of
other undetected cases," Dr. Paul Brown, former medical director of the National
Institutes of Health's Laboratory for Central Nervous System Studies and an
expert on mad cow-like diseases, told United Press International. "The question
was, 'How many?' and we still can't answer that."
Brown, who is preparing a scientific paper based on the latest two mad cow
cases to estimate the maximum number of infected cows that occurred in the
United States, said he has "absolutely no confidence in USDA tests before one
year ago" because of the agency's reluctance to retest the Texas cow that
initially tested positive.
USDA officials finally retested the cow and confirmed it was infected seven
months later, but only at the insistence of the agency's inspector general.
"Everything they did on the Texas cow makes everything USDA did before 2005
suspect," Brown said. ...snip...end
CDC - Bovine Spongiform Encephalopathy and Variant Creutzfeldt ... Dr. Paul
Brown is Senior Research Scientist in the Laboratory of Central Nervous System
... Address for correspondence: Paul Brown, Building 36, Room 4A-05, ...
PAUL BROWN COMMENT TO ME ON THIS ISSUE
Tuesday, September 12, 2006 11:10 AM
"Actually, Terry, I have been critical of the USDA handling of the mad cow
issue for some years, and with Linda Detwiler and others sent lengthy detailed
critiques and recommendations to both the USDA and the Canadian Food Agency."
OR, what the Honorable Phyllis Fong of the OIG found ;
Finding 2 Inherent Challenges in Identifying and Testing High-Risk Cattle
Still Remain
Table 1. Animal feed ingredients that are legally used in U.S. animal feeds
Animal
Rendered animal protein from Meat meal, meat meal tankage, meat and bone
meal, poultry meal, animal the slaughter of food by-product meal, dried animal
blood, blood meal, feather meal, egg-shell production animals and other meal,
hydrolyzed whole poultry, hydrolyzed hair, bone marrow, and animal animals
digest from dead, dying, diseased, or disabled animals including deer and elk
Animal waste Dried ruminant waste, dried swine waste, dried poultry litter, and
undried processed animal waste products
snip...
Conclusions
Food-animal production in the United States has changed markedly in the
past century, and these changes have paralleled major changes in animal feed
formulations. While this industrialized system of food-animal production may
result in increased production efficiencies, some of the changes in animal
feeding practices may result in unintended adverse health consequences for
consumers of animal-based food products. Currently, the use of animal feed
ingredients, including rendered animal products, animal waste, antibiotics,
metals, and fats, could result in higher levels of bacteria, antibioticresistant
bacteria, prions, arsenic, and dioxinlike compounds in animals and resulting
animal-based food products intended for human consumption. Subsequent human
health effects among consumers could include increases in bacterial infections
(antibioticresistant and nonresistant) and increases in the risk of developing
chronic (often fatal) diseases such as vCJD. Nevertheless, in spite of the wide
range of potential human health impacts that could result from animal feeding
practices, there are little data collected at the federal or state level
concerning the amounts of specific ingredients that are intentionally included
in U.S. animal feed. In addition, almost no biological or chemical testing is
conducted on complete U.S. animal feeds; insufficient testing is performed on
retail meat products; and human health effects data are not appropriately linked
to this information. These surveillance inadequacies make it difficult to
conduct rigorous epidemiologic studies and risk assessments that could identify
the extent to which specific human health risks are ultimately associated with
animal feeding practices. For example, as noted above, there are insufficient
data to determine whether other human foodborne bacterial illnesses besides
those caused by S. enterica serotype Agona are associated with animal feeding
practices. Likewise, there are insufficient data to determine the percentage of
antibiotic-resistant human bacterial infections that are attributed to the
nontherapeutic use of antibiotics in animal feed. Moreover, little research has
been conducted to determine whether the use of organoarsenicals in animal feed,
which can lead to elevated levels of arsenic in meat products (Lasky et al.
2004), contributes to increases in cancer risk. In order to address these
research gaps, the following principal actions are necessary within the United
States: a) implementation of a nationwide reporting system of the specific
amounts and types of feed ingredients of concern to public health that are
incorporated into animal feed, including antibiotics, arsenicals, rendered
animal products, fats, and animal waste; b) funding and development of robust
surveillance systems that monitor biological, chemical, and other etiologic
agents throughout the animal-based food-production chain “from farm to fork” to
human health outcomes; and c) increased communication and collaboration among
feed professionals, food-animal producers, and veterinary and public health
officials.
REFERENCES...snip...end
Sapkota et al. 668 VOLUME 115 | NUMBER 5 | May 2007 • Environmental Health
Perspectives
NOW, what about the product that is not reported ?
HOW many more Farm Fresh Meats, Inc. and Roland Emerson Farabee's are out
there that submitted bogus samples for the infamous June 2004 ENHANCED BSE MAD
COW COVER-UP, but did not get caught ?
IT was not bad enough to have the USDA et al bungle there own BSE Testing
Protocols up so bad, it took Weybridge and a year of hounding by s o m e, and
finally an act of Congress to finally get that cow confirmed, but once caught
there, i guess the next best thing would be to have bogus BSE testing samples
submitted for testing from healthy USDA cattle, what next ? not to forget about
the other stumbling and staggering Tejas mad cow they rendered without any test
at all, and the other Tejas mad cow that took 7+ months and an act of Congress
to finally get confirmed from Weybridge. my God, even the three stooges, laural
and hardy put all together could have thought up all this. $$$
The beef import forecast for the second quarter was unchanged from last
month’s, despite pressure from higher-than-expected domestic cow slaughter that
has remained high through most of this period. Beef imports into the United
States from Australia, New Zealand, and Uruguay provide additional processing
beef that, along with domestic cow and bull beef, is mixed with 50-percent trim
from fed cattle to make ground beef. Forecast beef exports were raised slightly,
mainly on continued (though gradual) improvements in sales to major Asian
markets. In late May the World Animal Health Organization – known by its French
acronym, OIE – designated the United States as having “controlled risk status”
for bovine spongiform encephalopathy, or BSE. This designation reflects the
OIE’s view that beef produced in the United States is safe for export, since BSE
control measures such as feed bans and removal of specified risk materials
result in negligible risk to consumers. However, the OIE standards are only
guidelines. Individual countries may adopt differing standards, and those
countries that do accept OIE standards must still undertake the bureaucratic
processes to revise their rules and procedures.
(Adopted by the International Committee of the OIE on 23 May 2006)
11. Information published by the OIE is derived from appropriate
declarations made by the official Veterinary Services of Member Countries. The
OIE is not responsible for inaccurate publication of country disease status
based on inaccurate information or changes in epidemiological status or other
significant events that were not promptly reported to then Central
Bureau............
P.S. Thank You Honorable Phyllis Fong for trying to keep them straight
anyway. ...TSS
Thursday, July 24, 2014
Protocol for further laboratory investigations into the distribution of
infectivity of Atypical BSE SCIENTIFIC REPORT OF EFSA
Saturday, June 12, 2010
PUBLICATION REQUEST AND FOIA REQUEST Project Number: 3625-32000-086-05
Study of Atypical Bse
infamous august 4, 1997 BSE TSE prion mad cow feed ban, part of usda fda et
al TRIPLE MAD COW FIREWALL, 10 YEARS AFTER ;
10,000,000+ LBS. of PROHIBITED BANNED MAD COW FEED I.E. BLOOD LACED MBM IN
COMMERCE USA 2007
Date: March 21, 2007 at 2:27 pm PST RECALLS AND FIELD CORRECTIONS:
VETERINARY MEDICINES -- CLASS II PRODUCT Bulk cattle feed made with recalled
Darling's 85% Blood Meal, Flash Dried, Recall # V-024-2007 CODE Cattle feed
delivered between 01/12/2007 and 01/26/2007 RECALLING FIRM/MANUFACTURER
Pfeiffer, Arno, Inc, Greenbush, WI. by conversation on February 5, 2007.
Firm initiated recall is ongoing. REASON Blood meal used to make cattle
feed was recalled because it was cross- contaminated with prohibited bovine meat
and bone meal that had been manufactured on common equipment and labeling did
not bear cautionary BSE statement.
VOLUME OF PRODUCT IN COMMERCE 42,090 lbs. DISTRIBUTION WI
___________________________________
PRODUCT Custom dairy premix products: MNM ALL PURPOSE Pellet, HILLSIDE/CDL
Prot- Buffer Meal, LEE, M.-CLOSE UP PX Pellet, HIGH DESERT/ GHC LACT Meal,
TATARKA, M CUST PROT Meal, SUNRIDGE/CDL PROTEIN Blend, LOURENZO, K PVM DAIRY
Meal, DOUBLE B DAIRY/GHC LAC Mineral, WEST PIONT/GHC CLOSEUP Mineral, WEST
POINT/GHC LACT Meal, JENKS, J/COMPASS PROTEIN Meal, COPPINI - 8# SPECIAL DAIRY
Mix, GULICK, L-LACT Meal (Bulk), TRIPLE J - PROTEIN/LACTATION, ROCK CREEK/GHC
MILK Mineral, BETTENCOURT/GHC S.SIDE MK-MN, BETTENCOURT #1/GHC MILK MINR,
V&C DAIRY/GHC LACT Meal, VEENSTRA, F/GHC LACT Meal, SMUTNY, A- BYPASS ML
W/SMARTA, Recall # V-025-2007 CODE The firm does not utilize a code - only
shipping documentation with commodity and weights identified. RECALLING
FIRM/MANUFACTURER Rangen, Inc, Buhl, ID, by letters on February 13 and 14, 2007.
Firm initiated recall is complete. REASON Products manufactured from bulk
feed containing blood meal that was cross contaminated with prohibited meat and
bone meal and the labeling did not bear cautionary BSE statement.
VOLUME OF PRODUCT IN COMMERCE 9,997,976 lbs. DISTRIBUTION ID and NV
END OF ENFORCEMENT REPORT FOR MARCH 21, 2007
*** Singeltary reply ; Molecular, Biochemical and Genetic Characteristics
of BSE in Canada Singeltary reply ;
infamous august 4, 1997 BSE TSE prion mad cow feed ban, part of usda fda et
al TRIPLE MAD COW FIREWALL, 16 YEARS AFTER ;
Sunday, December 15, 2013
FDA PART 589 -- SUBSTANCES PROHIBITED FROM USE IN ANIMAL FOOD OR FEED
VIOLATIONS OFFICIAL ACTION INDICATED OAI UPDATE DECEMBER 2013 UPDATE
SUMMARY REPORT CALIFORNIA ATYPICAL L-TYPE BOVINE SPONGIFORM ENCEPHALOPATHY
CASE INVESTIGATION JULY 2012 CALIFORNIA
Summary Report BSE 2012
Executive Summary
Saturday, August 4, 2012
Final Feed Investigation Summary - California atypical L-type BSE Case -
July 2012
Saturday, August 4, 2012
Update from APHIS Regarding Release of the Final Report on the BSE
Epidemiological Investigation
LET'S take a closer look at this new prionpathy or prionopathy, and then
let's look at the g-h-BSEalabama mad cow. This new prionopathy in humans? the
genetic makeup is IDENTICAL to the g-h-BSEalabama mad cow, the only _documented_
mad cow in the world to date like this, ......wait, it get's better. this new
prionpathy is killing young and old humans, with LONG DURATION from onset of
symptoms to death, and the symptoms are very similar to nvCJD victims, OH, and
the plaques are very similar in some cases too, bbbut, it's not related to the
g-h-BSEalabama cow, WAIT NOW, it gets even better, the new human prionpathy that
they claim is a genetic TSE, has no relation to any gene mutation in that
family. daaa, ya think it could be related to that mad cow with the same genetic
make-up ??? there were literally tons and tons of banned mad cow protein in
Alabama in commerce, and none of it transmitted to cows, and the cows to humans
there from ??? r i g h t $$$ ALABAMA MAD COW g-h-BSEalabama In this study, we
identified a novel mutation in the bovine prion protein gene (Prnp), called
E211K, of a confirmed BSE positive cow from Alabama, United States of America.
This mutation is identical to the E200K pathogenic mutation found in humans with
a genetic form of CJD. This finding represents the first report of a confirmed
case of BSE with a potential pathogenic mutation within the bovine Prnp gene. We
hypothesize that the bovine Prnp E211K mutation most likely has caused BSE in
"the approximately 10-year-old cow" carrying the E221K mutation.
Saturday, August 14, 2010
BSE Case Associated with Prion Protein Gene Mutation (g-h-BSEalabama) and
VPSPr PRIONPATHY (see mad cow feed in COMMERCE IN ALABAMA...TSS)
her healthy calf also carried the mutation
(J. A. Richt and S. M. Hall PLoS Pathog. 4, e1000156; 2008).
This raises the possibility that the disease could occasionally be genetic
in origin. Indeed, the report of the UK BSE Inquiry in 2000 suggested that the
UK epidemic had most likely originated from such a mutation and argued against
the scrapierelated assumption. Such rare potential pathogenic PRNP mutations
could occur in countries at present considered to be free of BSE, such as
Australia and New Zealand. So it is important to maintain strict surveillance
for BSE in cattle, with rigorous enforcement of the ruminant feed ban (many
countries still feed ruminant proteins to pigs). Removal of specified risk
material, such as brain and spinal cord, from cattle at slaughter prevents
infected material from entering the human food chain. Routine genetic screening
of cattle for PRNP mutations, which is now available, could provide additional
data on the risk to the public. Because the point mutation identified in the
Alabama animals is identical to that responsible for the commonest type of
familial (genetic) CJD in humans, it is possible that the resulting infective
prion protein might cross the bovine-human species barrier more easily. Patients
with vCJD continue to be identified. The fact that this is happening less often
should not lead to relaxation of the controls necessary to prevent future
outbreaks.
Malcolm A. Ferguson-Smith Cambridge University Department of Veterinary
Medicine, Madingley Road, Cambridge CB3 0ES, UK e-mail: maf12@cam.ac.uk Jürgen
A. Richt College of Veterinary Medicine, Kansas State University, K224B Mosier
Hall, Manhattan, Kansas 66506-5601, USA NATURE|Vol 457|26 February 2009
Owens, Julie
From: Terry S. Singeltary Sr. [flounder9@verizon.net]
Sent: Monday, July 24, 2006 1:09 PM
To: FSIS RegulationsComments
Subject: [Docket No. FSIS-2006-0011] FSIS Harvard Risk Assessment of Bovine
Spongiform Encephalopathy (BSE) Page 1 of 98
FSIS, USDA, REPLY TO SINGELTARY
Thursday, November 28, 2013
Department of Justice Former Suppliers of Beef to National School Lunch
Program Settle Allegations of Improper Practices and Mistreating Cows
seems USDA NSLP et al thought that it would be alright, to feed our
children all across the USA, via the NSLP, DEAD STOCK DOWNER COWS, the most high
risk cattle for mad cow type disease, and other dangerous pathogens, and they
did this for 4 years, that was documented, then hid what they did by having a
recall, one of the largest recalls ever, and they made this recall and masked
the reason for the recall due to animal abuse (I do not condone animal abuse),
not for the reason of the potential for these animals to have mad cow BSE type
disease (or other dangerous and deadly pathogens). these TSE prion disease can
lay dormant for 5, 10, 20 years, or longer, WHO WILL WATCH OUR CHILDREN FOR THE
NEXT 5 DECADES FOR CJD ???
Saturday, September 21, 2013
Westland/Hallmark: 2008 Beef Recall A Case Study by The Food Industry
Center January 2010 THE FLIM-FLAM REPORT
DID YOUR CHILD CONSUME SOME OF THESE DEAD STOCK DOWNER COWS, THE MOST HIGH
RISK FOR MAD COW DISEASE ??? this recall was not for the welfare of the animals.
...tss you can check and see here ; (link now dead, does not work...tss)
try this link ;
Sunday, November 13, 2011
*** California BSE mad cow beef recall, QFC, CJD, and dead stock downer
livestock
Thursday, February 13, 2014
HSUS VS USDA ET AL BAN DOWNER CALVES FOR HUMAN CONSUMPTION (*veal) and
potential BSE risk factor there from
Transmissible Spongiform Encephalopathy TSE Prion Disease North America
2014
Transmissible Spongiform Encephalopathy TSE Prion Disease have now been
discovered in a wide verity of species across North America. typical C-BSE,
atypical L-type BASE BSE, atypical H-type BSE, atypical H-G BSE, of the bovine,
typical and atypical Scrapie strains, in sheep and goats, with atypical Nor-98
Scrapie spreading coast to coast in about 5 years. Chronic Wasting Disease CWD
in cervid is slowly spreading without any stopping it in Canada and the USA and
now has mutated into many different strains. Transmissible Mink Encephalopathy
TME outbreaks. These Transmissible Spongiform Encephalopathy TSE Prion Disease
have been silently mutating and spreading in different species in North America
for decades.
The USDA, FDA, et al have assured us of a robust Triple BSE TSE prion
Firewall, of which we now know without a doubt, that it was nothing but ink on
paper. Since the 1997 mad cow feed ban in the USA, literally tons and tons of
banned mad cow feed has been put out into commerce, never to return, as late as
December of 2013, serious, serious breaches in the FDA mad cow feed ban have
been documented. The 2004 enhanced BSE surveillance program was so flawed, that
one of the top TSE prion Scientist for the CDC, Dr. Paul Brown stated ; Brown,
who is preparing a scientific paper based on the latest two mad cow cases to
estimate the maximum number of infected cows that occurred in the United States,
said he has "absolutely no confidence in USDA tests before one year ago" because
of the agency's reluctance to retest the Texas cow that initially tested
positive.
see ;
The BSE surveillance and testing have also been proven to be flawed, and
the GAO and OIG have both raised serious question as to just how flawed it has
been (see GAO and OIG reports). North America has more documented TSE prion
disease, in different documented species (excluding the Zoo BSE animals in the
EU), then any other place on the Globe. This does not include the very
likelihood that TSE prion disease in the domestic feline and canine have been
exposed to high doses of the TSE prion disease vid pet food. To date, it’s still
legal to include deer from cwd zone into pet food or deer food. Specified Risk
Material i.e. SRM bans still being breach, as recently as just last month.
nvCJD or what they now call vCJD, another case documented in Texas last
month, with very little information being released to the public on about this
case? with still the same line of thought from federal officials, ‘it can’t
happen here’, so another vCJD blamed on travel of a foreign animal disease from
another country, while ignoring all the BSE TSE Prion risk factors we have here
in the USA and Canada, and the time that this victim and others, do spend in the
USA, and exposed to these risk factors, apparently do not count in any way with
regard to risk factor. a flawed process of risk assessment.
sporadic CJD, along with new TSE prion disease in humans, of which the
young are dying, of which long duration of illness from onset of symptoms to
death have been documented, only to have a new name added to the pot of prion
disease i.e. sporadic GSS, sporadic FFI, and or VPSPR. I only ponder how a
familial type disease could be sporadic with no genetic link to any family
member? when the USA is the only documented Country in the world to have
documented two different cases of atypical H-type BSE, with one case being
called atypical H-G BSE with the G meaning Genetic, with new science now showing
that indeed atypical H-type BSE is very possible transmitted to cattle via oral
transmission (Prion2014). sporadic CJD and VPSPR have been rising in Canada,
USA, and the UK, with the same old excuse, better surveillance. You can only use
that excuse for so many years, for so many decades, until one must conclude that
CJD TSE prion cases are rising. a 48% incease in CJD in Canada is not just a
blip or a reason of better surveillance, it is a mathematical rise in numbers.
More and more we are seeing more humans exposed in various circumstance in the
Hospital, Medical, Surgical arenas to the TSE Prion disease, and at the same
time in North America, more and more humans are becoming exposed to the TSE
prion disease via consumption of the TSE prion via deer and elk, cattle, sheep
and goats, and for those that are exposed via or consumption, go on to further
expose many others via the iatrogenic modes of transmission of the TSE prion
disease i.e. friendly fire. I pondered this mode of transmission via the victims
of sporadic FFI, sporadic GSS, could this be a iatrogenic event from someone
sub-clinical with sFFI or sGSS ? what if?
Two decades have passed since Dr. Ironside first confirmed his first ten
nvCJD victims in 1995. Ten years later, 2005, we had Dr. Gambetti and his first
ten i.e. VPSPR in younger victims. now we know that indeed VPSPR is
transmissible. yet all these TSE prion disease and victims in the USA and Canada
are being pawned off as a spontaneous event, yet science has shown, the
spontaneous theory has never been proven in any natural case of TSE prion
disease, and scientist have warned, that they have now linked some sporadic CJD
cases to atypical BSE, to atypical Scrapie, and to CWD, yet we don’t here about
this in the public domain. We must make all human and animal TSE prion disease
reportable in every age group, in ever state and internationally, we must have a
serious re-evaluation and testing of the USA cattle herds, and we must ban
interstate movement of all cervids. Any voluntary effort to do any of this will
fail. Folks, we have let the industry run science far too long with regards to
the TSE prion disease. While the industry and their lobbyist continues to funnel
junk science to our decision policy makers, Rome burns. ...end
REFERENCES
Sunday, June 29, 2014
Transmissible Spongiform Encephalopathy TSE Prion Disease North America
2014
Saturday, June 14, 2014
Rep. Rosa DeLauro (D-CT) Calls for Briefing on Beef Recalled for Mad Cow
Potential Rep. Rosa DeLauro (D-CT)
Thursday, June 12, 2014
Missouri Firm Recalls Ribeye and Carcass Products That May Contain
Specified Risk Materials 4,012 pounds of fresh beef products because the dorsal
root ganglia may not have been completely removed
Monday, July 28, 2014
Mitigating the Risk of Transmission and Environmental Contamination of
Transmissible Spongiform Encephalopathies 2013 Annual Report
Tuesday, August 12, 2014
MAD COW USDA TSE PRION COVER UP or JUST IGNORANCE, for the record AUGUST
2014
***********CJD REPORT 1994 increased risk for consumption of veal and
venison and lamb***********
CREUTZFELDT JAKOB DISEASE SURVEILLANCE IN THE UNITED KINGDOM THIRD ANNUAL
REPORT AUGUST 1994
Consumption of venison and veal was much less widespread among both cases
and controls. For both of these meats there was evidence of a trend with
increasing frequency of consumption being associated with increasing risk of
CJD. (not nvCJD, but sporadic CJD...tss)
These associations were largely unchanged when attention was restricted to
pairs with data obtained from relatives. ...
Table 9 presents the results of an analysis of these data.
There is STRONG evidence of an association between ‘’regular’’ veal eating
and risk of CJD (p = .0.01).
Individuals reported to eat veal on average at least once a year appear to
be at 13 TIMES THE RISK of individuals who have never eaten veal.
There is, however, a very wide confidence interval around this estimate.
There is no strong evidence that eating veal less than once per year is
associated with increased risk of CJD (p = 0.51).
The association between venison eating and risk of CJD shows similar
pattern, with regular venison eating associated with a 9 FOLD INCREASE IN RISK
OF CJD (p = 0.04).
There is some evidence that risk of CJD INCREASES WITH INCREASING FREQUENCY
OF LAMB EATING (p = 0.02).
The evidence for such an association between beef eating and CJD is weaker
(p = 0.14). When only controls for whom a relative was interviewed are included,
this evidence becomes a little STRONGER (p = 0.08).
snip...
It was found that when veal was included in the model with another
exposure, the association between veal and CJD remained statistically
significant (p = < 0.05 for all exposures), while the other exposures ceased
to be statistically significant (p = > 0.05).
snip...
In conclusion, an analysis of dietary histories revealed statistical
associations between various meats/animal products and INCREASED RISK OF CJD.
When some account was taken of possible confounding, the association between
VEAL EATING AND RISK OF CJD EMERGED AS THE STRONGEST OF THESE ASSOCIATIONS
STATISTICALLY. ...
snip...
In the study in the USA, a range of foodstuffs were associated with an
increased risk of CJD, including liver consumption which was associated with an
apparent SIX-FOLD INCREASE IN THE RISK OF CJD. By comparing the data from 3
studies in relation to this particular dietary factor, the risk of liver
consumption became non-significant with an odds ratio of 1.2 (PERSONAL
COMMUNICATION, PROFESSOR A. HOFMAN. ERASMUS UNIVERSITY, ROTTERDAM). (???...TSS)
snip...see full report ;
Thursday, October 10, 2013
*************CJD REPORT 1994 increased risk for consumption of veal and
venison and lamb**************
PEO752/2 0097
CONFIDENTIAL-POLICY MARKET SENSITIVE-LIMITED DISTRIBUTION
BSE AND CJD
RECEIVED
25 MAR 1996
BSE AND CJD
The Prime Minister held a meeting on Tuesday 19 March to discuss the latest
scientific information on Bovine Spongiform Encephalopathy (BSE) and Creutzfeldt
Jacob Disease (CJD), The Deputy Prime Minister, the Lord President, Chief
Secretary, Lord Privy Seal, the Secretary of State for Health, the Secretary of
State for Scotland, the Minister for Agriculture. the Financial Secretary, the
Attorney General, the Minister for Food, the Chief Whip, Sir Robin Butler, Keith
Meldrum (Chief Veterinary Officer), Professor Pattison (Chairman of SEAC), Dr
Eileen Rubery (Department of Health), Richard Packer (MAFF), Lord McColl, John
Ward, Howell James, Alex Allan, Jonathan Haslam, Robert Culpin (HM Treasury),
Kenneth Mackenzie (Cabinet Office), Tim Sutton (HM Treasury) were also present.
The Deputy Prime Minister, the Chief Secretary to the Treasury and the Lord
Privy Seal all announced that they had relevant interests in the form of cattle
herds.
The Prime Minister began the meeting by commenting that some very difficult
decisions needed to be taken to ensure that the correct balance was struck
between treating this matter seriously and over-reacting. Colleagues needed to
recall that there were many issues which remained unknown.
Professor Pattison said that his committee had considered the new
information which had become available very carefully and had examined in detail
all the possible options. ***The situation was that there were now nine cases of
CJD which appeared to be different from classical CJD. There were, in addition,
three other possibJe cases. The cases tended to be among the young but varied
from those aged 18 to age 41. The new variant CJD showed. an atypical clinical
picture with an unknown pathology. This had persuaded SEAC that the variant was
distinct. No cases in the UK or abroad had been seen before which matched to
this pattern. The Committee had considered the new methods of monitoring the
occurrence of the disease and were aware lab techniques had improved, but they
bad drawn the conclusion that they could not persuade themselves that it was
more careful observation alone which had brought these cases to light.
This implied that there might be a new risk factor and in the view of the
Committee the most likely explanation was that BSE was that risk factor. To date
however the evidence was not available which proved that BSE could be linked to
these cases. It appeared to the Committee to be the most likely explanation but
they might be wrong. It might, for example, be that the new form had always been
present in a low incidence but had remained unreported or there-might be an
entirely separate new environmental factor. However, the committee was of the
view that the most likely cause was something new in the cattle population in
the mid-1980s which was causing something new in the human population in the
mid-1990s. This was in their view likely to be exposure to BSE before the
introduction of the SBO controls.
Professor Pattison noted that it was impossible to predict how many more
cases there might be and it might well be eighteen months before the full extent
of the problem could be ascertained. A dozen or so might be the limit or it
might remain at a relatively low level as in cats and unlike in the cattle
population it had not escalated. The cattle epidemic with escalating numbers was
probably due to feeding cattle remains back to cattle. This had not happened
with cats nor of course with humans.
The committee believed it was increasingly impossible to keep this
information confidential. Members of the Committee had already had to attend two
expert meetings where they had not been able to provide colleagues with the full
story. Given the increasingly high risk of a leak it was the Committee's view
that a controlled statement by the Government would be more appropriate. The
Committee had considered whether extra restrictions on human consumption of beef
or beef products would be necessary. They had not concluded that immediate
measures were necessary other than to stress the importance of implementing
existing controls as nearly perfectly as possible. The Committee would consider
again at the weekend what more might be done. ranging from a do nothing option
to the slaughter of the national herd.
Personally. Professor Pattison did not think that extreme measures would be
necessary. In his view the committee was more likely to focus on controls
concerning older cattle, together with further controls on mechanically
recovered meat. ...snip...end...tss
96/03.19/16.2
CONFIDENTIAL – POLICY MARKET SENSITIVE - LIMITED DISTRIBUTION
CONFIDENTIAL – POLICY
SEAC
Since there were indications that the news was about to break, there was no
reason to prevent all members of the Committee joining the meeting in
London...
Sunday, July 06, 2014
Dietary Risk Factors for Sporadic Creutzfeldt-Jakob Disease: A Confirmatory
Case-Control Study
Conclusions—The a priori hypotheses were supported.
*Consumption of various meat products may be one method of transmission of
the infectious agent for sCJD.
Seven main threats for the future linked to prions
***Also, a link is suspected between atypical BSE and some apparently
sporadic cases of Creutzfeldt-Jakob disease in humans. These atypical BSE cases
constitute an unforeseen first threat that could sharply modify the European
approach to prion diseases.
Second threat
snip...
Monday, October 10, 2011
EFSA Journal 2011 The European Response to BSE: A Success Story
snip...
*** but the possibility that a small proportion of human cases so far
classified as "sporadic" CJD are of zoonotic origin could not be excluded.
Moreover, transmission experiments to non-human primates suggest that some TSE
agents in addition to Classical BSE prions in cattle (namely L-type Atypical
BSE, Classical BSE in sheep, transmissible mink encephalopathy (TME) and chronic
wasting disease (CWD) agents) might have zoonotic potential.
snip...
***In addition, non-human primates are specifically susceptible for
atypical BSE as demonstrated by an approximately 50% shortened incubation time
for L-type BSE as compared to C-type. Considering the current scientific
information available, it cannot be assumed that these different BSE types pose
the same human health risks as C-type BSE or that these risks are mitigated by
the same protective measures.
***Infectivity in skeletal muscle of BASE-infected cattle
***feedstuffs- It also suggests a similar cause or source for atypical BSE
in these countries.
Friday, December 5, 2014
*** SPECIAL ALERT The OIE recommends strengthening animal disease
surveillance worldwide ***
OIE BSE TSE PRION AKA MAD COW DISEASE ?
‘’the silence was deafening’’ ...tss
Wednesday, December 3, 2014
Over 200 Groups Urge Congress to Continue Supporting COOL
For Immediate Release
Tuesday, December 2, 2014
*** UK EXPORTS OF MBM TO WORLD Bovine Spongiform Encephalopathy BSE TSE
Prion aka Mad Cow Disease
USA, NORTH AMERICA, MBM (or any potential TSE prion disease) EXPORTS TO THE
WORLD (?) [protected by the BSE MRR policy] $$$
Monday, December 1, 2014
Germany Bovine Spongiform Encephalopathy BSE CJD TSE Prion disease A Review
December 1, 2014
Friday, November 28, 2014
BOVINE SPONGIFORM ENCEPHALOPATHY BSE AKA MAD COW DISEASE PORTUGAL CONFIRMED
Sunday, October 5, 2014
France stops BSE testing for Mad Cow Disease
Monday, May 5, 2014
Brazil BSE Mad Cow disease confirmed OIE 02/05/2014
BSE INQUIRY DFAs
Sunday, May 18, 2008
BSE Inquiry DRAFT FACTUAL ACCOUNT DFA
BSE Inquiry DRAFT FACTUAL ACCOUNTS DFA's
Sunday, May 18, 2008
***BSE, CJD, and Baby foods (the great debate 1999 to 2005)
Sunday, May 18, 2008
***MAD COW DISEASE BSE CJD CHILDREN VACCINES
Sunday, December 7, 2014
Scientific update on the potential for transmissibility of non-prion
protein misfolding diseases PRIONOIDS
Saturday, December 13, 2014
Terry S. Singeltary Sr. Publications TSE prion disease
for my files...tss
Diagnosis and Reporting of Creutzfeldt-Jakob Disease
Singeltary, Sr et al. JAMA.2001; 285: 733-734. Vol. 285 No. 6, February 14,
2001 JAMA
Sunday, November 23, 2014
Confirmed Variant Creutzfeldt-Jakob Disease (variant CJD) Case in Texas in
June 2014 confirmed as USA case NOT European
‘’The specific overseas country where this patient’s infection occurred is
less clear largely because the investigation did not definitely link him to a
country where other known vCJD cases likely had been infected.’’
layperson
just made a promise, never forget, never let them forget...
MOM DOD 12/14/97 confirmed hvCJD Heidenhain Variant Creutzfeldt Jakob
Disease Case Report
snip...
Heidenhain Variant Creutzfeldt Jakob Disease autopsy case report 'MOM'
DIVISION OF NEUROPATHOLOGY University of Texas Medical Branch 114
McCullough Bldg. Galveston, Texas 77555-0785 FAX COVER SHEET DATE: 4-23-98 TO:
Mr. Terry Singeltary @ ------- FROM: Gerald Campbell FAX: (409) 772-5315 PHONE:
(409) 772-2881 Number of Pages (including cover sheet): Message:
*CONFIDENTIALITY NOTICE* This document accompanying this transmission contains
confidential information belonging to the sender that is legally privileged.
This information is intended only for the use of the individual or entry names
above. If you are not the intended recipient, you are hereby notified that any
disclosure, copying distribution, or the taking of any action in reliances on
the contents of this telefaxed information is strictly prohibited. If you
received this telefax in error, please notify us by telephone immediately to
arrange for return of the original documents.
--------------------------
Patient Account: 90000014-518 Med. Rec. No.: (0160)118511Q
Patient Name: POULTER, BARBARA
Age: 63
YRS DOB: 10/17/34
Sex: F
Admitting Race: C
Attending Dr.: Date / Time Admitted : 12/14/97 1228
Copies to: UTMB University of Texas Medical Branch Galveston, Texas
77555-0543 (409) 772-1238 Fax (409) 772-5683 Pathology Report
FINAL AUTOPSY DIAGNOSIS
Autopsy' Office (409)772-2858 Autopsy NO.: AU-97-00435 AUTOPSY INFORMATION:
Occupation: Unknown Birthplace: Unknown Residence: Crystal Beach Date/Time
of Death: 12/14/97 13:30 Date/Time of Autopsy: 12/15/97 15:00
Pathologist/Resident: Pencil/Fernandez Service: Private Restriction:
Brain only FINAL AUTOPSY DIAGNOSIS I. Brain: Creutzfeldt-Jakob disease,
Heidenhain variant.
snip...see full text ;
well, it’s been 17 years to the day.
just made a promise to Mom, DOD December 14, 1997 confirmed hvCJD, never
forget, and never let them forget...
Terry S. Singeltary Sr.
Sunday, December 14, 2014
ALERT new variant Creutzfeldt Jakob Disease nvCJD or vCJD, sporadic CJD
strains, TSE prion aka Mad Cow Disease United States of America Update December
14, 2014 Report
TSS
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