Friday, December 19, 2014

Rancho Alleged Cancerous Eyeball Case Going To Trial

Rancho Co-Owner Will Go To Trial Alone, Three Others Make Plea Deals


By Dan Flynn | December 19, 2014


 Jury selection will begin July 16, 2015, in the federal criminal conspiracy case involving former Rancho Feeding Corp. co-owner Jesse J. Amaral Jr. The 76-year-old cattle company executive will be tried alone as three others, implicated in the alleged conspiracy to sell for human consumption cattle known to have cancerous eyeballs, have all made deals with the prosecution.


 Felix Sandoval Cabrera, 55, the foreman of Rancho’s slaughterhouse at Petaluma, CA, is the latest to reach a plea agreement with the government, entering a single guilty plea to count 7 of the original indictment last Aug. 14 charging him with distribution of adulterated, misbranded and uninspected meat.


 Earlier, Eugene D. Corda, the 65-year-old Rancho yardman, and 77-year-old Robert Singleton, Rancho’s other co-owner, also entered guilty pleas to the same sole count.


 That leaves only Amaral going to trial next July. The court-approved plea agreements the other three defendants have with the government are sealed, but it’s likely that all three have agreed to appear at the trial as government witnesses against him.


 The prosecution has charged the former Rancho co-owner with 11 federal felony counts. The government will attempt to prove to a jury that the Petaluma resident is guilty of one count of conspiracy to distribute adulterated, misbranded and uninspected meat, two counts of conspiracy to commit mail fraud, six counts of distribution of adulterated and misbranded meat, one count of conspiracy to commit mail fraud, and two counts of mail fraud.


 If convicted on all counts, Amaral could be sentenced to more than 100 years in jail and fined more than $1.3 million.


 Proceedings for all the defendants are being held before U.S. District Judge Charles R. Breyer in San Francisco. He has scheduled a status conference for Singleton on Feb. 18, 2015, and another for Cabrera and Corda on Aug. 12, 2015, which will likely be after the Amaral trial has concluded.


 Amaral, Rancho’s president and general manager, was in control of the day-to-day operations at the Petaluma slaughterhouse located 60 miles north of San Francisco. Singleton’s role was to buy cattle and supervise processed beef for distribution.


 Cabrera, Rancho’s foreman, was responsible for the staff and the “kill floor,” including being responsible for “knocking cattle,” or stunning them immediately prior to slaughter.


 As yardman, Corda was responsible for receiving cattle and moving them to the proper areas for inspection and slaughter.


 Singleton bought cattle from both auction houses and individual farmers and ranchers in Northern California and Nevada.


 “Some of the purchased cattle exhibited signs of epithelioma, that is lumps or other abnormalities around the eye, and were less expensive than cattle that appeared completely healthy,” according to the indictment.


 When cattle with these eye conditions arrived at the Petaluma slaughterhouse, Corda or another Rancho employee would allegedly place them in pen 9A, court documents state. The owners were in charge of determining the order in which cattle were processed for inspection and slaughter.


 When instructed, Corda moved cattle into the pen designated for ante-mortem inspection by the USDA veterinarian or Food Safety and Inspection Service (FSIS) personnel. Cattle passing ante-mortem inspection generally went immediately into the kill chute, where they were knocked, slaughtered and inspected again post mortem. After passing the post-mortem inspection, the carcass was tagged and could be sold.


 In mid-2012, Amaral is accused of ordering Rancho employees to process cattle that were condemned by the USDA veterinarian. At his instruction, Cabrera allegedly had workers cut the “USDA Condemned” stamps out of the cattle carcasses so they could be processed for sale and distribution.


 At about the same time, prosecutors say Amaral gave the foreman, Cabrera, and the yardman, Corda, directions on how to circumvent inspection procedures for cows with cancerous eyes. Both Amaral and Singleton allegedly told their employees to swap out uninspected cows with cancerous eyes with cattle that had already passed ante-mortem inspection.


 “Cabrera knocked the cancer eye cows, and he or another kill floor employee at his instruction slaughtered them and deposited their heads in the gut bin,” the indictment states. “Cabrera, or another kill floor employee at his instruction, placed heads from apparently healthy cows, which had been previously reserved, next to the cancer eye cow carcasses.


 “The switch and slaughter of un-inspected cancer eye cows occurred during the inspectors’ lunch breaks, at a time during which plant operations were supposed to cease,” the charging documents continue. “When the inspectors returned from lunch for post mortem inspections, they were unaware that the carcasses they were inspecting belonged to cancer eye cows that had escaped ante mortem inspection.”


 Based on Rancho’s records, the government found that, from January 2013 to January 2014, beef from 101 head of condemned cattle and 79 cows with eye cancer was processed for human consumption. For every diseased animal that he got past USDA inspectors, Cabrera allegedly got a $50 bonus.


 In early 2014, with the federal investigation underway, Rancho recalled all of its beef production going back the previous year, or about 8.7 million pounds. It also was forced to close down and ultimately sell the Petaluma slaughterhouse. The facility has since resumed operations under new ownership.


 © Food Safety News


More Headlines from Food Policy & Law »


Tags: Eugene D. Corda, Felix Sandoval Cabrera, Jesse J. Amaral Jr., Petaluma, Rancho, Rancho Feeding Corporation, Robert Singleton








see more on Rancho history here;


Friday, March 21, 2014


Rancho Dead Stock Cancer Downers Recall Explained FSIS March 20 2014 ?


“As of March 20, 2014, FSIS has completed all checks (effectiveness checks and disposition verification checks) for recalls 002-2014 and 013-2014 regarding Rancho Feeding Corporation. FSIS has determined that based on the number of successful checks (see Directive 8080.1, Attachment 1, Table 3) where businesses were notified of the recall and removed affected products from commerce that the recall activities were effective.”



Thursday, March 20, 2014





Thursday, March 6, 2014


TEXAS RECALL LIST MASSIVE FROM DEAD STOCK DOWNER CANCER COWS OFFAL from Class I Recall 002-2014 and 013-2014 Health Risk: High Jan 13, 2014 and Feb 8, 2014 shipped to Texas, Florida, and Illinois UPDATE FEBRUARY 14, 2014



Thursday, February 27, 2014





>>>“Cabrera knocked the cancer eye cows, and he or another kill floor employee at his instruction slaughtered them and deposited their heads in the gut bin,” the indictment states. “Cabrera, or another kill floor employee at his instruction, placed heads from apparently healthy cows, which had been previously reserved, next to the cancer eye cow carcasses. <<<


where have I heard something similar before ?

Thursday, November 18, 2010



Galen Niehues, an inspector for the Nebraska Department of Agriculture, (NDA), was convicted of mail fraud for submitting falsified reports to his employer concerning inspections he was supposed to perform at Nebraska cattle operations. Niehues was tasked with performing inspections of Nebraska ranches, cattle and feed for the presence of neurological diseases in cattle including Bovine Spongiform Encephalopathy (BSE), also known as “Mad Cow Disease”. Niehues was to identify cattle producers, perform on-site inspections of the farm sites and cattle operations, ask producers specific questions about feed, and take samples of the feed. Niehues was to then submit feed samples for laboratory analysis, and complete reports of his inspections and submit them to the NDA and to the Federal Food and Drug Administration (FDA). An investigation by the FDA and NDA revealed Niehues had fabricated approximately 100 BSE inspections and inspection reports. When confronted, Niehues admitted his reports were fraudulent, and that had fabricated the reports and feed samples he submitted to the NDA. Niehues received a sentence of 5 years probation, a 3-year term of supervised release, and was required to pay $42,812.10 in restitution.




Date: June 21, 2007 at 2:49 pm PST


Owner and Corporation Plead Guilty to Defrauding Bovine Spongiform Encephalopathy (BSE) Surveillance Program


An Arizona meat processing company and its owner pled guilty in February 2007 to charges of theft of Government funds, mail fraud, and wire fraud. The owner and his company defrauded the BSE Surveillance Program when they falsified BSE Surveillance Data Collection Forms and then submitted payment requests to USDA for the services. In addition to the targeted sample population (those cattle that were more than 30 months old or had other risk factors for BSE), the owner submitted to USDA, or caused to be submitted, BSE obex (brain stem) samples from healthy USDA-inspected cattle. As a result, the owner fraudulently received approximately $390,000. Sentencing is scheduled for May 2007.




Topics that will be covered in ongoing or planned reviews under Goal 1 include:


soundness of BSE maintenance sampling (APHIS),


implementation of Performance-Based Inspection System enhancements for specified risk material (SRM) violations and improved inspection controls over SRMs (FSIS and APHIS),




The findings and recommendations from these efforts will be covered in future semiannual reports as the relevant audits and investigations are completed.





-MORE Office of the United States Attorney District of Arizona


FOR IMMEDIATE RELEASE For Information Contact Public Affairs


February 16, 2007 WYN HORNBUCKLE Telephone: (602) 514-7625 Cell: (602) 525-2681




PHOENIX -- Farm Fresh Meats, Inc. and Roland Emerson Farabee, 55, of Maricopa, Arizona, pleaded guilty to stealing $390,000 in government funds, mail fraud and wire fraud, in federal district court in Phoenix. U.S. Attorney Daniel Knauss stated, “The integrity of the system that tests for mad cow disease relies upon the honest cooperation of enterprises like Farm Fresh Meats. Without that honest cooperation, consumers both in the U.S. and internationally are at risk. We want to thank the USDA’s Office of Inspector General for their continuing efforts to safeguard the public health and enforce the law.” Farm Fresh Meats and Farabee were charged by Information with theft of government funds, mail fraud and wire fraud. According to the Information, on June 7, 2004, Farabee, on behalf of Farm Fresh Meats, signed a contract with the U.S. Department of Agriculture (the “USDA Agreement”) to collect obex samples from cattle at high risk of mad cow disease (the “Targeted Cattle Population”). The Targeted Cattle Population consisted of the following cattle: cattle over thirty months of age; nonambulatory cattle; cattle exhibiting signs of central nervous system disorders; cattle exhibiting signs of mad cow disease; and dead cattle. Pursuant to the USDA Agreement, the USDA agreed to pay Farm Fresh Meats $150 per obex sample for collecting obex samples from cattle within the Targeted Cattle Population, and submitting the obex samples to a USDA laboratory for mad cow disease testing. Farm Fresh Meats further agreed to maintain in cold storage the sampled cattle carcasses and heads until the test results were received by Farm Fresh Meats.


Evidence uncovered during the government’s investigation established that Farm Fresh Meats and Farabee submitted samples from cattle outside the Targeted Cattle Population. Specifically, Farm Fresh Meats and Farabee submitted, or caused to be submitted, obex samples from healthy, USDA inspected cattle, in order to steal government moneys.


Evidence collected also demonstrated that Farm Fresh Meats and Farabee failed to maintain cattle carcasses and heads pending test results and falsified corporate books and records to conceal their malfeasance. Such actions, to the extent an obex sample tested positive (fortunately, none did), could have jeopardized the USDA’s ability to identify the diseased animal and pinpoint its place of origin. On Wednesday, February 14, 2007, Farm Fresh Meats and Farabee pleaded guilty to stealing government funds and using the mails and wires to effect the scheme. According to their guilty pleas:


(a) Farm Fresh Meats collected, and Farabee directed others to collect, obex samples from cattle outside the Targeted Cattle Population, which were not subject to payment by the USDA;


(b) Farm Fresh Meats 2 and Farabee caused to be submitted payment requests to the USDA knowing that the requests were based on obex samples that were not subject to payment under the USDA Agreement;


(c) Farm Fresh Meats completed and submitted, and Farabee directed others to complete and submit, BSE Surveillance Data Collection Forms to the USDA’s testing laboratory that were false and misleading;


(d) Farm Fresh Meats completed and submitted, and Farabee directed others to complete and submit, BSE Surveillance Submission Forms filed with the USDA that were false and misleading;


(e) Farm Fresh Meats falsified, and Farabee directed others to falsify, internal Farm Fresh Meats documents to conceal the fact that Farm Fresh Meats was seeking and obtaining payment from the USDA for obex samples obtained from cattle outside the Targeted Cattle Population; and


(f) Farm Fresh Meats failed to comply with, and Farabee directed others to fail to comply with, the USDA Agreement by discarding cattle carcasses and heads prior to receiving BSE test results. A conviction for theft of government funds carries a maximum penalty of 10 years imprisonment. Mail fraud and wire fraud convictions carry a maximum penalty of 20 years imprisonment. Convictions for the above referenced violations also carry a maximum fine of $250,000 for individuals and $500,000 for organizations. In determining an actual sentence, Judge Earl H. Carroll will consult the U.S. Sentencing Guidelines, which provide appropriate sentencing ranges. The judge, however, is not bound by those guidelines in determining a sentence.


Sentencing is set before Judge Earl H. Carroll on May 14, 2007. The investigation in this case was conducted by Assistant Special Agent in Charge Alejandro Quintero, United States Department of Agriculture, Office of Inspector General. The prosecution is being handled by Robert Long, Assistant U.S. Attorney, District of Arizona, Phoenix. CASE NUMBER: CR-07-00160-PHX-EHC RELEASE NUMBER: 2007-051(Farabee) # # #



WE can only hope that this is a single incident. BUT i have my doubts. I remember when the infamous TOKEN Purina Feed Mill in Texas was feeding up to 5.5 grams of potentially and probably tainted BANNED RUMINANT feed to cattle, and the FDA was bragging at the time that the amount of potentially BANNED product was so little and the cattle were so big ;


"It is important to note that the prohibited material was domestic in origin (therefore not likely to contain infected material because there is no evidence of BSE in U.S. cattle), fed at a very low level, and fed only once. The potential risk of BSE to such cattle is therefore exceedingly low, even if the feed were contaminated."



On Friday, April 30 th , the Food and Drug Administration learned that a cow with central nervous system symptoms had been killed and shipped to a processor for rendering into animal protein for use in animal feed. ... FDA's investigation showed that the animal in question had already been rendered into "meat and bone meal" (a type of protein animal feed). Over the weekend FDA was able to track down all the implicated material. That material is being held by the firm, which is cooperating fully with FDA.



WE now know all that was a lie. WE know that literally Thousands of TONS of BANNED and most likely tainted product is still going out to commerce. WE know now and we knew then that .005 to a gram was lethal. WE know that CWD infected deer and elk, scrapie infected sheep, BSE and BASE infected cattle have all been rendered and fed back to livestock (including cattle) for human and animal consumption.


Paul Brown, known and respected TSE scientist, former TSE expert for the CDC said he had ''absolutely no confidence in USDA tests before one year ago'', and this was on March 15, 2006 ;


"The fact the Texas cow showed up fairly clearly implied the existence of other undetected cases," Dr. Paul Brown, former medical director of the National Institutes of Health's Laboratory for Central Nervous System Studies and an expert on mad cow-like diseases, told United Press International. "The question was, 'How many?' and we still can't answer that."


Brown, who is preparing a scientific paper based on the latest two mad cow cases to estimate the maximum number of infected cows that occurred in the United States, said he has "absolutely no confidence in USDA tests before one year ago" because of the agency's reluctance to retest the Texas cow that initially tested positive.


USDA officials finally retested the cow and confirmed it was infected seven months later, but only at the insistence of the agency's inspector general.


"Everything they did on the Texas cow makes everything USDA did before 2005 suspect," Brown said. ...snip...end



CDC - Bovine Spongiform Encephalopathy and Variant Creutzfeldt ... Dr. Paul Brown is Senior Research Scientist in the Laboratory of Central Nervous System ... Address for correspondence: Paul Brown, Building 36, Room 4A-05, ...





Tuesday, September 12, 2006 11:10 AM


"Actually, Terry, I have been critical of the USDA handling of the mad cow issue for some years, and with Linda Detwiler and others sent lengthy detailed critiques and recommendations to both the USDA and the Canadian Food Agency."


OR, what the Honorable Phyllis Fong of the OIG found ;


Finding 2 Inherent Challenges in Identifying and Testing High-Risk Cattle Still Remain



Table 1. Animal feed ingredients that are legally used in U.S. animal feeds




Rendered animal protein from Meat meal, meat meal tankage, meat and bone meal, poultry meal, animal the slaughter of food by-product meal, dried animal blood, blood meal, feather meal, egg-shell production animals and other meal, hydrolyzed whole poultry, hydrolyzed hair, bone marrow, and animal animals digest from dead, dying, diseased, or disabled animals including deer and elk Animal waste Dried ruminant waste, dried swine waste, dried poultry litter, and undried processed animal waste products






Food-animal production in the United States has changed markedly in the past century, and these changes have paralleled major changes in animal feed formulations. While this industrialized system of food-animal production may result in increased production efficiencies, some of the changes in animal feeding practices may result in unintended adverse health consequences for consumers of animal-based food products. Currently, the use of animal feed ingredients, including rendered animal products, animal waste, antibiotics, metals, and fats, could result in higher levels of bacteria, antibioticresistant bacteria, prions, arsenic, and dioxinlike compounds in animals and resulting animal-based food products intended for human consumption. Subsequent human health effects among consumers could include increases in bacterial infections (antibioticresistant and nonresistant) and increases in the risk of developing chronic (often fatal) diseases such as vCJD. Nevertheless, in spite of the wide range of potential human health impacts that could result from animal feeding practices, there are little data collected at the federal or state level concerning the amounts of specific ingredients that are intentionally included in U.S. animal feed. In addition, almost no biological or chemical testing is conducted on complete U.S. animal feeds; insufficient testing is performed on retail meat products; and human health effects data are not appropriately linked to this information. These surveillance inadequacies make it difficult to conduct rigorous epidemiologic studies and risk assessments that could identify the extent to which specific human health risks are ultimately associated with animal feeding practices. For example, as noted above, there are insufficient data to determine whether other human foodborne bacterial illnesses besides those caused by S. enterica serotype Agona are associated with animal feeding practices. Likewise, there are insufficient data to determine the percentage of antibiotic-resistant human bacterial infections that are attributed to the nontherapeutic use of antibiotics in animal feed. Moreover, little research has been conducted to determine whether the use of organoarsenicals in animal feed, which can lead to elevated levels of arsenic in meat products (Lasky et al. 2004), contributes to increases in cancer risk. In order to address these research gaps, the following principal actions are necessary within the United States: a) implementation of a nationwide reporting system of the specific amounts and types of feed ingredients of concern to public health that are incorporated into animal feed, including antibiotics, arsenicals, rendered animal products, fats, and animal waste; b) funding and development of robust surveillance systems that monitor biological, chemical, and other etiologic agents throughout the animal-based food-production chain “from farm to fork” to human health outcomes; and c) increased communication and collaboration among feed professionals, food-animal producers, and veterinary and public health officials.




Sapkota et al. 668 VOLUME 115 | NUMBER 5 | May 2007 • Environmental Health Perspectives



NOW, what about the product that is not reported ?


HOW many more Farm Fresh Meats, Inc. and Roland Emerson Farabee's are out there that submitted bogus samples for the infamous June 2004 ENHANCED BSE MAD COW COVER-UP, but did not get caught ?


IT was not bad enough to have the USDA et al bungle there own BSE Testing Protocols up so bad, it took Weybridge and a year of hounding by s o m e, and finally an act of Congress to finally get that cow confirmed, but once caught there, i guess the next best thing would be to have bogus BSE testing samples submitted for testing from healthy USDA cattle, what next ? not to forget about the other stumbling and staggering Tejas mad cow they rendered without any test at all, and the other Tejas mad cow that took 7+ months and an act of Congress to finally get confirmed from Weybridge. my God, even the three stooges, laural and hardy put all together could have thought up all this. $$$


The beef import forecast for the second quarter was unchanged from last month’s, despite pressure from higher-than-expected domestic cow slaughter that has remained high through most of this period. Beef imports into the United States from Australia, New Zealand, and Uruguay provide additional processing beef that, along with domestic cow and bull beef, is mixed with 50-percent trim from fed cattle to make ground beef. Forecast beef exports were raised slightly, mainly on continued (though gradual) improvements in sales to major Asian markets. In late May the World Animal Health Organization – known by its French acronym, OIE – designated the United States as having “controlled risk status” for bovine spongiform encephalopathy, or BSE. This designation reflects the OIE’s view that beef produced in the United States is safe for export, since BSE control measures such as feed bans and removal of specified risk materials result in negligible risk to consumers. However, the OIE standards are only guidelines. Individual countries may adopt differing standards, and those countries that do accept OIE standards must still undertake the bureaucratic processes to revise their rules and procedures.



(Adopted by the International Committee of the OIE on 23 May 2006)


11. Information published by the OIE is derived from appropriate declarations made by the official Veterinary Services of Member Countries. The OIE is not responsible for inaccurate publication of country disease status based on inaccurate information or changes in epidemiological status or other significant events that were not promptly reported to then Central Bureau............



P.S. Thank You Honorable Phyllis Fong for trying to keep them straight anyway. ...TSS


Thursday, July 24, 2014


Protocol for further laboratory investigations into the distribution of infectivity of Atypical BSE SCIENTIFIC REPORT OF EFSA



Saturday, June 12, 2010


PUBLICATION REQUEST AND FOIA REQUEST Project Number: 3625-32000-086-05 Study of Atypical Bse



infamous august 4, 1997 BSE TSE prion mad cow feed ban, part of usda fda et al TRIPLE MAD COW FIREWALL, 10 YEARS AFTER ;




Date: March 21, 2007 at 2:27 pm PST RECALLS AND FIELD CORRECTIONS: VETERINARY MEDICINES -- CLASS II PRODUCT Bulk cattle feed made with recalled Darling's 85% Blood Meal, Flash Dried, Recall # V-024-2007 CODE Cattle feed delivered between 01/12/2007 and 01/26/2007 RECALLING FIRM/MANUFACTURER Pfeiffer, Arno, Inc, Greenbush, WI. by conversation on February 5, 2007.


Firm initiated recall is ongoing. REASON Blood meal used to make cattle feed was recalled because it was cross- contaminated with prohibited bovine meat and bone meal that had been manufactured on common equipment and labeling did not bear cautionary BSE statement.






PRODUCT Custom dairy premix products: MNM ALL PURPOSE Pellet, HILLSIDE/CDL Prot- Buffer Meal, LEE, M.-CLOSE UP PX Pellet, HIGH DESERT/ GHC LACT Meal, TATARKA, M CUST PROT Meal, SUNRIDGE/CDL PROTEIN Blend, LOURENZO, K PVM DAIRY Meal, DOUBLE B DAIRY/GHC LAC Mineral, WEST PIONT/GHC CLOSEUP Mineral, WEST POINT/GHC LACT Meal, JENKS, J/COMPASS PROTEIN Meal, COPPINI - 8# SPECIAL DAIRY Mix, GULICK, L-LACT Meal (Bulk), TRIPLE J - PROTEIN/LACTATION, ROCK CREEK/GHC MILK Mineral, BETTENCOURT/GHC S.SIDE MK-MN, BETTENCOURT #1/GHC MILK MINR, V&C DAIRY/GHC LACT Meal, VEENSTRA, F/GHC LACT Meal, SMUTNY, A- BYPASS ML W/SMARTA, Recall # V-025-2007 CODE The firm does not utilize a code - only shipping documentation with commodity and weights identified. RECALLING FIRM/MANUFACTURER Rangen, Inc, Buhl, ID, by letters on February 13 and 14, 2007.


Firm initiated recall is complete. REASON Products manufactured from bulk feed containing blood meal that was cross contaminated with prohibited meat and bone meal and the labeling did not bear cautionary BSE statement.







*** Singeltary reply ; Molecular, Biochemical and Genetic Characteristics of BSE in Canada Singeltary reply ;



infamous august 4, 1997 BSE TSE prion mad cow feed ban, part of usda fda et al TRIPLE MAD COW FIREWALL, 16 YEARS AFTER ;


Sunday, December 15, 2013







Summary Report BSE 2012


Executive Summary



Saturday, August 4, 2012


Final Feed Investigation Summary - California atypical L-type BSE Case - July 2012



Saturday, August 4, 2012


Update from APHIS Regarding Release of the Final Report on the BSE Epidemiological Investigation



LET'S take a closer look at this new prionpathy or prionopathy, and then let's look at the g-h-BSEalabama mad cow. This new prionopathy in humans? the genetic makeup is IDENTICAL to the g-h-BSEalabama mad cow, the only _documented_ mad cow in the world to date like this, ......wait, it get's better. this new prionpathy is killing young and old humans, with LONG DURATION from onset of symptoms to death, and the symptoms are very similar to nvCJD victims, OH, and the plaques are very similar in some cases too, bbbut, it's not related to the g-h-BSEalabama cow, WAIT NOW, it gets even better, the new human prionpathy that they claim is a genetic TSE, has no relation to any gene mutation in that family. daaa, ya think it could be related to that mad cow with the same genetic make-up ??? there were literally tons and tons of banned mad cow protein in Alabama in commerce, and none of it transmitted to cows, and the cows to humans there from ??? r i g h t $$$ ALABAMA MAD COW g-h-BSEalabama In this study, we identified a novel mutation in the bovine prion protein gene (Prnp), called E211K, of a confirmed BSE positive cow from Alabama, United States of America. This mutation is identical to the E200K pathogenic mutation found in humans with a genetic form of CJD. This finding represents the first report of a confirmed case of BSE with a potential pathogenic mutation within the bovine Prnp gene. We hypothesize that the bovine Prnp E211K mutation most likely has caused BSE in "the approximately 10-year-old cow" carrying the E221K mutation.




Saturday, August 14, 2010


BSE Case Associated with Prion Protein Gene Mutation (g-h-BSEalabama) and VPSPr PRIONPATHY (see mad cow feed in COMMERCE IN ALABAMA...TSS)



her healthy calf also carried the mutation


(J. A. Richt and S. M. Hall PLoS Pathog. 4, e1000156; 2008).


This raises the possibility that the disease could occasionally be genetic in origin. Indeed, the report of the UK BSE Inquiry in 2000 suggested that the UK epidemic had most likely originated from such a mutation and argued against the scrapierelated assumption. Such rare potential pathogenic PRNP mutations could occur in countries at present considered to be free of BSE, such as Australia and New Zealand. So it is important to maintain strict surveillance for BSE in cattle, with rigorous enforcement of the ruminant feed ban (many countries still feed ruminant proteins to pigs). Removal of specified risk material, such as brain and spinal cord, from cattle at slaughter prevents infected material from entering the human food chain. Routine genetic screening of cattle for PRNP mutations, which is now available, could provide additional data on the risk to the public. Because the point mutation identified in the Alabama animals is identical to that responsible for the commonest type of familial (genetic) CJD in humans, it is possible that the resulting infective prion protein might cross the bovine-human species barrier more easily. Patients with vCJD continue to be identified. The fact that this is happening less often should not lead to relaxation of the controls necessary to prevent future outbreaks.


Malcolm A. Ferguson-Smith Cambridge University Department of Veterinary Medicine, Madingley Road, Cambridge CB3 0ES, UK e-mail: Jürgen A. Richt College of Veterinary Medicine, Kansas State University, K224B Mosier Hall, Manhattan, Kansas 66506-5601, USA NATURE|Vol 457|26 February 2009



Owens, Julie


From: Terry S. Singeltary Sr. []


Sent: Monday, July 24, 2006 1:09 PM


To: FSIS RegulationsComments


Subject: [Docket No. FSIS-2006-0011] FSIS Harvard Risk Assessment of Bovine Spongiform Encephalopathy (BSE) Page 1 of 98






Thursday, November 28, 2013


Department of Justice Former Suppliers of Beef to National School Lunch Program Settle Allegations of Improper Practices and Mistreating Cows



seems USDA NSLP et al thought that it would be alright, to feed our children all across the USA, via the NSLP, DEAD STOCK DOWNER COWS, the most high risk cattle for mad cow type disease, and other dangerous pathogens, and they did this for 4 years, that was documented, then hid what they did by having a recall, one of the largest recalls ever, and they made this recall and masked the reason for the recall due to animal abuse (I do not condone animal abuse), not for the reason of the potential for these animals to have mad cow BSE type disease (or other dangerous and deadly pathogens). these TSE prion disease can lay dormant for 5, 10, 20 years, or longer, WHO WILL WATCH OUR CHILDREN FOR THE NEXT 5 DECADES FOR CJD ???


Saturday, September 21, 2013


Westland/Hallmark: 2008 Beef Recall A Case Study by The Food Industry Center January 2010 THE FLIM-FLAM REPORT



DID YOUR CHILD CONSUME SOME OF THESE DEAD STOCK DOWNER COWS, THE MOST HIGH RISK FOR MAD COW DISEASE ??? this recall was not for the welfare of the animals. ...tss you can check and see here ; (link now dead, does not work...tss)



try this link ;



Sunday, November 13, 2011


*** California BSE mad cow beef recall, QFC, CJD, and dead stock downer livestock



Thursday, February 13, 2014


HSUS VS USDA ET AL BAN DOWNER CALVES FOR HUMAN CONSUMPTION (*veal) and potential BSE risk factor there from



Transmissible Spongiform Encephalopathy TSE Prion Disease North America 2014


Transmissible Spongiform Encephalopathy TSE Prion Disease have now been discovered in a wide verity of species across North America. typical C-BSE, atypical L-type BASE BSE, atypical H-type BSE, atypical H-G BSE, of the bovine, typical and atypical Scrapie strains, in sheep and goats, with atypical Nor-98 Scrapie spreading coast to coast in about 5 years. Chronic Wasting Disease CWD in cervid is slowly spreading without any stopping it in Canada and the USA and now has mutated into many different strains. Transmissible Mink Encephalopathy TME outbreaks. These Transmissible Spongiform Encephalopathy TSE Prion Disease have been silently mutating and spreading in different species in North America for decades.


The USDA, FDA, et al have assured us of a robust Triple BSE TSE prion Firewall, of which we now know without a doubt, that it was nothing but ink on paper. Since the 1997 mad cow feed ban in the USA, literally tons and tons of banned mad cow feed has been put out into commerce, never to return, as late as December of 2013, serious, serious breaches in the FDA mad cow feed ban have been documented. The 2004 enhanced BSE surveillance program was so flawed, that one of the top TSE prion Scientist for the CDC, Dr. Paul Brown stated ; Brown, who is preparing a scientific paper based on the latest two mad cow cases to estimate the maximum number of infected cows that occurred in the United States, said he has "absolutely no confidence in USDA tests before one year ago" because of the agency's reluctance to retest the Texas cow that initially tested positive.


see ;



The BSE surveillance and testing have also been proven to be flawed, and the GAO and OIG have both raised serious question as to just how flawed it has been (see GAO and OIG reports). North America has more documented TSE prion disease, in different documented species (excluding the Zoo BSE animals in the EU), then any other place on the Globe. This does not include the very likelihood that TSE prion disease in the domestic feline and canine have been exposed to high doses of the TSE prion disease vid pet food. To date, it’s still legal to include deer from cwd zone into pet food or deer food. Specified Risk Material i.e. SRM bans still being breach, as recently as just last month.


nvCJD or what they now call vCJD, another case documented in Texas last month, with very little information being released to the public on about this case? with still the same line of thought from federal officials, ‘it can’t happen here’, so another vCJD blamed on travel of a foreign animal disease from another country, while ignoring all the BSE TSE Prion risk factors we have here in the USA and Canada, and the time that this victim and others, do spend in the USA, and exposed to these risk factors, apparently do not count in any way with regard to risk factor. a flawed process of risk assessment.


sporadic CJD, along with new TSE prion disease in humans, of which the young are dying, of which long duration of illness from onset of symptoms to death have been documented, only to have a new name added to the pot of prion disease i.e. sporadic GSS, sporadic FFI, and or VPSPR. I only ponder how a familial type disease could be sporadic with no genetic link to any family member? when the USA is the only documented Country in the world to have documented two different cases of atypical H-type BSE, with one case being called atypical H-G BSE with the G meaning Genetic, with new science now showing that indeed atypical H-type BSE is very possible transmitted to cattle via oral transmission (Prion2014). sporadic CJD and VPSPR have been rising in Canada, USA, and the UK, with the same old excuse, better surveillance. You can only use that excuse for so many years, for so many decades, until one must conclude that CJD TSE prion cases are rising. a 48% incease in CJD in Canada is not just a blip or a reason of better surveillance, it is a mathematical rise in numbers. More and more we are seeing more humans exposed in various circumstance in the Hospital, Medical, Surgical arenas to the TSE Prion disease, and at the same time in North America, more and more humans are becoming exposed to the TSE prion disease via consumption of the TSE prion via deer and elk, cattle, sheep and goats, and for those that are exposed via or consumption, go on to further expose many others via the iatrogenic modes of transmission of the TSE prion disease i.e. friendly fire. I pondered this mode of transmission via the victims of sporadic FFI, sporadic GSS, could this be a iatrogenic event from someone sub-clinical with sFFI or sGSS ? what if?


Two decades have passed since Dr. Ironside first confirmed his first ten nvCJD victims in 1995. Ten years later, 2005, we had Dr. Gambetti and his first ten i.e. VPSPR in younger victims. now we know that indeed VPSPR is transmissible. yet all these TSE prion disease and victims in the USA and Canada are being pawned off as a spontaneous event, yet science has shown, the spontaneous theory has never been proven in any natural case of TSE prion disease, and scientist have warned, that they have now linked some sporadic CJD cases to atypical BSE, to atypical Scrapie, and to CWD, yet we don’t here about this in the public domain. We must make all human and animal TSE prion disease reportable in every age group, in ever state and internationally, we must have a serious re-evaluation and testing of the USA cattle herds, and we must ban interstate movement of all cervids. Any voluntary effort to do any of this will fail. Folks, we have let the industry run science far too long with regards to the TSE prion disease. While the industry and their lobbyist continues to funnel junk science to our decision policy makers, Rome burns. ...end




Sunday, June 29, 2014


Transmissible Spongiform Encephalopathy TSE Prion Disease North America 2014



Saturday, June 14, 2014


Rep. Rosa DeLauro (D-CT) Calls for Briefing on Beef Recalled for Mad Cow Potential Rep. Rosa DeLauro (D-CT)



Thursday, June 12, 2014


Missouri Firm Recalls Ribeye and Carcass Products That May Contain Specified Risk Materials 4,012 pounds of fresh beef products because the dorsal root ganglia may not have been completely removed



Monday, July 28, 2014


Mitigating the Risk of Transmission and Environmental Contamination of Transmissible Spongiform Encephalopathies 2013 Annual Report



Tuesday, August 12, 2014





***********CJD REPORT 1994 increased risk for consumption of veal and venison and lamb***********




Consumption of venison and veal was much less widespread among both cases and controls. For both of these meats there was evidence of a trend with increasing frequency of consumption being associated with increasing risk of CJD. (not nvCJD, but sporadic CJD...tss)


These associations were largely unchanged when attention was restricted to pairs with data obtained from relatives. ...


Table 9 presents the results of an analysis of these data.


There is STRONG evidence of an association between ‘’regular’’ veal eating and risk of CJD (p = .0.01).


Individuals reported to eat veal on average at least once a year appear to be at 13 TIMES THE RISK of individuals who have never eaten veal.


There is, however, a very wide confidence interval around this estimate. There is no strong evidence that eating veal less than once per year is associated with increased risk of CJD (p = 0.51).


The association between venison eating and risk of CJD shows similar pattern, with regular venison eating associated with a 9 FOLD INCREASE IN RISK OF CJD (p = 0.04).


There is some evidence that risk of CJD INCREASES WITH INCREASING FREQUENCY OF LAMB EATING (p = 0.02).


The evidence for such an association between beef eating and CJD is weaker (p = 0.14). When only controls for whom a relative was interviewed are included, this evidence becomes a little STRONGER (p = 0.08).




It was found that when veal was included in the model with another exposure, the association between veal and CJD remained statistically significant (p = < 0.05 for all exposures), while the other exposures ceased to be statistically significant (p = > 0.05).




In conclusion, an analysis of dietary histories revealed statistical associations between various meats/animal products and INCREASED RISK OF CJD. When some account was taken of possible confounding, the association between VEAL EATING AND RISK OF CJD EMERGED AS THE STRONGEST OF THESE ASSOCIATIONS STATISTICALLY. ...




In the study in the USA, a range of foodstuffs were associated with an increased risk of CJD, including liver consumption which was associated with an apparent SIX-FOLD INCREASE IN THE RISK OF CJD. By comparing the data from 3 studies in relation to this particular dietary factor, the risk of liver consumption became non-significant with an odds ratio of 1.2 (PERSONAL COMMUNICATION, PROFESSOR A. HOFMAN. ERASMUS UNIVERSITY, ROTTERDAM). (???...TSS)


snip...see full report ;



Thursday, October 10, 2013


*************CJD REPORT 1994 increased risk for consumption of veal and venison and lamb**************



PEO752/2 0097








25 MAR 1996




The Prime Minister held a meeting on Tuesday 19 March to discuss the latest scientific information on Bovine Spongiform Encephalopathy (BSE) and Creutzfeldt Jacob Disease (CJD), The Deputy Prime Minister, the Lord President, Chief Secretary, Lord Privy Seal, the Secretary of State for Health, the Secretary of State for Scotland, the Minister for Agriculture. the Financial Secretary, the Attorney General, the Minister for Food, the Chief Whip, Sir Robin Butler, Keith Meldrum (Chief Veterinary Officer), Professor Pattison (Chairman of SEAC), Dr Eileen Rubery (Department of Health), Richard Packer (MAFF), Lord McColl, John Ward, Howell James, Alex Allan, Jonathan Haslam, Robert Culpin (HM Treasury), Kenneth Mackenzie (Cabinet Office), Tim Sutton (HM Treasury) were also present.


The Deputy Prime Minister, the Chief Secretary to the Treasury and the Lord Privy Seal all announced that they had relevant interests in the form of cattle herds.


The Prime Minister began the meeting by commenting that some very difficult decisions needed to be taken to ensure that the correct balance was struck between treating this matter seriously and over-reacting. Colleagues needed to recall that there were many issues which remained unknown.


Professor Pattison said that his committee had considered the new information which had become available very carefully and had examined in detail all the possible options. ***The situation was that there were now nine cases of CJD which appeared to be different from classical CJD. There were, in addition, three other possibJe cases. The cases tended to be among the young but varied from those aged 18 to age 41. The new variant CJD showed. an atypical clinical picture with an unknown pathology. This had persuaded SEAC that the variant was distinct. No cases in the UK or abroad had been seen before which matched to this pattern. The Committee had considered the new methods of monitoring the occurrence of the disease and were aware lab techniques had improved, but they bad drawn the conclusion that they could not persuade themselves that it was more careful observation alone which had brought these cases to light.


This implied that there might be a new risk factor and in the view of the Committee the most likely explanation was that BSE was that risk factor. To date however the evidence was not available which proved that BSE could be linked to these cases. It appeared to the Committee to be the most likely explanation but they might be wrong. It might, for example, be that the new form had always been present in a low incidence but had remained unreported or there-might be an entirely separate new environmental factor. However, the committee was of the view that the most likely cause was something new in the cattle population in the mid-1980s which was causing something new in the human population in the mid-1990s. This was in their view likely to be exposure to BSE before the introduction of the SBO controls.


Professor Pattison noted that it was impossible to predict how many more cases there might be and it might well be eighteen months before the full extent of the problem could be ascertained. A dozen or so might be the limit or it might remain at a relatively low level as in cats and unlike in the cattle population it had not escalated. The cattle epidemic with escalating numbers was probably due to feeding cattle remains back to cattle. This had not happened with cats nor of course with humans.


The committee believed it was increasingly impossible to keep this information confidential. Members of the Committee had already had to attend two expert meetings where they had not been able to provide colleagues with the full story. Given the increasingly high risk of a leak it was the Committee's view that a controlled statement by the Government would be more appropriate. The Committee had considered whether extra restrictions on human consumption of beef or beef products would be necessary. They had not concluded that immediate measures were necessary other than to stress the importance of implementing existing controls as nearly perfectly as possible. The Committee would consider again at the weekend what more might be done. ranging from a do nothing option to the slaughter of the national herd.


Personally. Professor Pattison did not think that extreme measures would be necessary. In his view the committee was more likely to focus on controls concerning older cattle, together with further controls on mechanically recovered meat. ...snip...end...tss











Since there were indications that the news was about to break, there was no reason to prevent all members of the Committee joining the meeting in London...



Sunday, July 06, 2014


Dietary Risk Factors for Sporadic Creutzfeldt-Jakob Disease: A Confirmatory Case-Control Study


Conclusions—The a priori hypotheses were supported.


*Consumption of various meat products may be one method of transmission of the infectious agent for sCJD.



Seven main threats for the future linked to prions


***Also, a link is suspected between atypical BSE and some apparently sporadic cases of Creutzfeldt-Jakob disease in humans. These atypical BSE cases constitute an unforeseen first threat that could sharply modify the European approach to prion diseases.


Second threat





Monday, October 10, 2011


EFSA Journal 2011 The European Response to BSE: A Success Story




*** but the possibility that a small proportion of human cases so far classified as "sporadic" CJD are of zoonotic origin could not be excluded. Moreover, transmission experiments to non-human primates suggest that some TSE agents in addition to Classical BSE prions in cattle (namely L-type Atypical BSE, Classical BSE in sheep, transmissible mink encephalopathy (TME) and chronic wasting disease (CWD) agents) might have zoonotic potential.






***In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures.



***Infectivity in skeletal muscle of BASE-infected cattle



***feedstuffs- It also suggests a similar cause or source for atypical BSE in these countries.



Friday, December 5, 2014


*** SPECIAL ALERT The OIE recommends strengthening animal disease surveillance worldwide ***




‘’the silence was deafening’’ ...tss



Wednesday, December 3, 2014


Over 200 Groups Urge Congress to Continue Supporting COOL


For Immediate Release



Tuesday, December 2, 2014


*** UK EXPORTS OF MBM TO WORLD Bovine Spongiform Encephalopathy BSE TSE Prion aka Mad Cow Disease


USA, NORTH AMERICA, MBM (or any potential TSE prion disease) EXPORTS TO THE WORLD (?) [protected by the BSE MRR policy] $$$



Monday, December 1, 2014


Germany Bovine Spongiform Encephalopathy BSE CJD TSE Prion disease A Review December 1, 2014



Friday, November 28, 2014





Sunday, October 5, 2014


France stops BSE testing for Mad Cow Disease



Monday, May 5, 2014


Brazil BSE Mad Cow disease confirmed OIE 02/05/2014






Sunday, May 18, 2008







Sunday, May 18, 2008


***BSE, CJD, and Baby foods (the great debate 1999 to 2005)



Sunday, May 18, 2008





Sunday, December 7, 2014


Scientific update on the potential for transmissibility of non-prion protein misfolding diseases PRIONOIDS



Saturday, December 13, 2014


Terry S. Singeltary Sr. Publications TSE prion disease


for my files...tss


Diagnosis and Reporting of Creutzfeldt-Jakob Disease


Singeltary, Sr et al. JAMA.2001; 285: 733-734. Vol. 285 No. 6, February 14, 2001 JAMA



Sunday, November 23, 2014


Confirmed Variant Creutzfeldt-Jakob Disease (variant CJD) Case in Texas in June 2014 confirmed as USA case NOT European


‘’The specific overseas country where this patient’s infection occurred is less clear largely because the investigation did not definitely link him to a country where other known vCJD cases likely had been infected.’’





just made a promise, never forget, never let them forget...


MOM DOD 12/14/97 confirmed hvCJD Heidenhain Variant Creutzfeldt Jakob Disease Case Report




Heidenhain Variant Creutzfeldt Jakob Disease autopsy case report 'MOM'


DIVISION OF NEUROPATHOLOGY University of Texas Medical Branch 114 McCullough Bldg. Galveston, Texas 77555-0785 FAX COVER SHEET DATE: 4-23-98 TO: Mr. Terry Singeltary @ ------- FROM: Gerald Campbell FAX: (409) 772-5315 PHONE: (409) 772-2881 Number of Pages (including cover sheet): Message: *CONFIDENTIALITY NOTICE* This document accompanying this transmission contains confidential information belonging to the sender that is legally privileged. This information is intended only for the use of the individual or entry names above. If you are not the intended recipient, you are hereby notified that any disclosure, copying distribution, or the taking of any action in reliances on the contents of this telefaxed information is strictly prohibited. If you received this telefax in error, please notify us by telephone immediately to arrange for return of the original documents.




Patient Account: 90000014-518 Med. Rec. No.: (0160)118511Q




Age: 63


YRS DOB: 10/17/34


Sex: F


Admitting Race: C


Attending Dr.: Date / Time Admitted : 12/14/97 1228


Copies to: UTMB University of Texas Medical Branch Galveston, Texas 77555-0543 (409) 772-1238 Fax (409) 772-5683 Pathology Report




Autopsy' Office (409)772-2858 Autopsy NO.: AU-97-00435 AUTOPSY INFORMATION:


Occupation: Unknown Birthplace: Unknown Residence: Crystal Beach Date/Time of Death: 12/14/97 13:30 Date/Time of Autopsy: 12/15/97 15:00 Pathologist/Resident: Pencil/Fernandez Service: Private Restriction:


Brain only FINAL AUTOPSY DIAGNOSIS I. Brain: Creutzfeldt-Jakob disease, Heidenhain variant.


snip...see full text ;




well, it’s been 17 years to the day.


just made a promise to Mom, DOD December 14, 1997 confirmed hvCJD, never forget, and never let them forget...


Terry S. Singeltary Sr.


Sunday, December 14, 2014


ALERT new variant Creutzfeldt Jakob Disease nvCJD or vCJD, sporadic CJD strains, TSE prion aka Mad Cow Disease United States of America Update December 14, 2014 Report




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